| Tumour necrosis factor α, lipid peroxidation and NO• are increased and associated with decreased free-radical scavenging enzymes in patients with Weill-Marchesani syndrome | |
Abstract/OtherAbstract
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AIM: Weill-Marchesani syndrome (WMS) is a rare systemic disorder with both autosomal recessive and dominant inheritances. Accumulation of reactive oxygen species such as O2⋅-, H2O2 and OH• causes lipid peroxidation (LPO), whereas antioxidant enzymes (superoxide dismutase (SOD), glutathione peroxidase (GSHPx)) mediate defence against oxidative stress. Excess tumour necrosis factor (TNF)-α and NO• react with O2⋅- and cause further antioxidant depletion with an increase in mutation frequency by H2O2. This study investigated the levels of SOD, GSHPx, catalase (CAT), TNF-α, NO• and LPO in patients with WMS. |
Authors
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Cem Evereklioglu, Yusuf Turkoz, Mustafa Calis, Fuat Duygulu, Aysun B. Karabulut |
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Contributors
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Publication Detail
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Publisher : Hindawi Publishing Corporation Type : - Format : - |
Date Detail
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1992 |
Subject
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Antioxidant enzymes, Lipid peroxidation, Nitric oxide, Tumour necrosis factor α, Weill-Marchesani syndrome. |
Coverage
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Relation
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Source
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Mediators of Inflammation |
Copyright Information
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Other Details
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Languages : eng |
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