Document Detail


A transcriptionally permissive epigenetic landscape at the vasoactive intestinal peptide receptor-1 promoter suggests a euchromatin nuclear position in murine CD4 T cells.
MedLine Citation:
PMID:  19729043     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
T cells express receptors for neuropeptides that mediate immunological activities. Vasoactive intestinal peptide receptor-1 (VPAC1), the prototypical group II G protein coupled receptor, binds two neuropeptides with high-affinity, called vasoactive intestinal peptide and pituitary adenylate cyclase activating polypeptide. During T cell signaling, VPAC1 mRNA expression levels are significantly downregulated through a Src kinase dependent mechanism, thus altering the sensitivity for these neuropeptides during an immune reaction. Presently, it is unknown whether the mechanism that regulates VPAC1 during T cell signaling involves epigenetic changes. Therefore, we hypothesized that the epigenetic landscape consisting of diacetylation at H3K9/14 and trimethylation at H3K4, two transcriptionally permissive histone modifications, would parallel VPAC1 expression showing high enrichment in untreated T cells, but lower enrichment in alpha-CD3 treated T cells. To this end, quantitative chromatin immunoprecipitation (ChIP) analysis of H3K9/14ac and H3K4me3 was conducted using purified CD4(+) T cells, with CD45R(+) B cells as a negative control. Our data revealed that these histone modifications at the VPAC1 promoter did indeed parallel its mRNA levels between T and B lymphocytes, but did not decrease during T cell signaling. Collectively, these data strongly imply a euchromatin nuclear position for the VPAC1 locus irrespective of the activation status of T cells.
Authors:
K D Benton; R J Hermann; E E Vomhof-DeKrey; J S Haring; T Van der Steen; J Smith; S Dovat; G P Dorsam
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2009-09-01
Journal Detail:
Title:  Regulatory peptides     Volume:  158     ISSN:  1873-1686     ISO Abbreviation:  Regul. Pept.     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-10-13     Completed Date:  2010-01-25     Revised Date:  2011-06-06    
Medline Journal Info:
Nlm Unique ID:  8100479     Medline TA:  Regul Pept     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  68-76     Citation Subset:  IM    
Affiliation:
Department of Chemistry and Molecular Biology, North Dakota State University, Fargo, ND 58108, USA.
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MeSH Terms
Descriptor/Qualifier:
Acetylation
Animals
Base Sequence
CD4-Positive T-Lymphocytes / metabolism*
Cell Nucleus / metabolism*
Chromatin Immunoprecipitation
DNA Primers
Down-Regulation
Epigenesis, Genetic*
Euchromatin / metabolism*
Methylation
Mice
Polymerase Chain Reaction
RNA, Messenger / genetics
Receptors, Vasoactive Intestinal Polypeptide, Type I / genetics*
Signal Transduction
Transcription, Genetic
Grant Support
ID/Acronym/Agency:
1KO1DK064828/DK/NIDDK NIH HHS; 2P20RR015566/RR/NCRR NIH HHS; K01 DK064828-05/DK/NIDDK NIH HHS; P20RR016741/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/DNA Primers; 0/Euchromatin; 0/RNA, Messenger; 0/Receptors, Vasoactive Intestinal Polypeptide, Type I

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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