Document Detail


The total cavopulmonary connection resistance: a significant impact on single ventricle hemodynamics at rest and exercise.
MedLine Citation:
PMID:  18931028     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Little is known about the impact of the total cavopulmonary connection (TCPC) on resting and exercise hemodynamics in a single ventricle (SV) circulation. The aim of this study was to elucidate this mechanism using a lumped parameter model of the SV circulation. Pulmonary vascular resistance (1.96+/-0.80 WU) and systemic vascular resistances (18.4+/-7.2 WU) were obtained from catheterization data on 40 patients with a TCPC. TCPC resistances (0.39+/-0.26 WU) were established using computational fluid dynamic simulations conducted on anatomically accurate three-dimensional models reconstructed from MRI (n=16). These parameters were used in a lumped parameter model of the SV circulation to investigate the impact of TCPC resistance on SV hemodynamics under resting and exercise conditions. A biventricular model was used for comparison. For a biventricular circulation, the cardiac output (CO) dependence on TCPC resistance was negligible (sensitivity=-0.064 l.min(-1).WU(-1)) but not for the SV circulation (sensitivity=-0.88 l.min(-1).WU(-1)). The capacity to increase CO with heart rate was also severely reduced for the SV. At a simulated heart rate of 150 beats/min, the SV patient with the highest resistance (1.08 WU) had a significantly lower increase in CO (20.5%) compared with the SV patient with the lowest resistance (50%) and normal circulation (119%). This was due to the increased afterload (+35%) and decreased preload (-12%) associated with the SV circulation. In conclusion, TCPC resistance has a significant impact on resting hemodynamics and the exercise capacity of patients with a SV physiology.
Authors:
Kartik S Sundareswaran; Kerem Pekkan; Lakshmi P Dasi; Kevin Whitehead; Shiva Sharma; Kirk R Kanter; Mark A Fogel; Ajit P Yoganathan
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Publication Detail:
Type:  Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2008-10-17
Journal Detail:
Title:  American journal of physiology. Heart and circulatory physiology     Volume:  295     ISSN:  0363-6135     ISO Abbreviation:  Am. J. Physiol. Heart Circ. Physiol.     Publication Date:  2008 Dec 
Date Detail:
Created Date:  2008-12-05     Completed Date:  2009-01-30     Revised Date:  2010-09-21    
Medline Journal Info:
Nlm Unique ID:  100901228     Medline TA:  Am J Physiol Heart Circ Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  H2427-35     Citation Subset:  IM    
Affiliation:
Wallace H. Coulter Department of Biomedical Engineering, School of Biomedical Engineering, Georgia Institute of Technology, Atlanta, GA 30332-0535, USA.
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Cardiac Output
Child
Child, Preschool
Computer Simulation
Databases as Topic
Exercise Tolerance*
Fontan Procedure*
Heart Catheterization
Heart Defects, Congenital / pathology,  physiopathology*,  surgery
Heart Rate
Hemodynamics*
Humans
Magnetic Resonance Angiography
Magnetic Resonance Imaging, Cine
Models, Anatomic
Models, Cardiovascular
Pulmonary Artery / pathology,  physiopathology*,  surgery
Pulmonary Circulation*
Reproducibility of Results
Treatment Outcome
United States
Vascular Resistance
Vena Cava, Inferior / pathology,  physiopathology*,  surgery
Vena Cava, Superior / pathology,  physiopathology*,  surgery
Ventricular Function*
Grant Support
ID/Acronym/Agency:
HL-67622/HL/NHLBI NIH HHS
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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