Document Detail


A CDE/CHR tandem element regulates cell cycle-dependent repression of cyclin B2 transcription.
MedLine Citation:
PMID:  11068036     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cyclin B is an important regulator of progression through the cell division cycle. The oscillating appearance of cyclin B1 and B2 proteins during the cell cycle is in part due to fluctuating mRNA levels. We had identified earlier a tandem promoter element named cell cycle-dependent element (CDE) and cell cycle genes homology region (CHR) which regulates cell cycle-dependent transcription of cdc25C, cyclin A and cdc2. Here we describe that cyclin B2 transcription is repressed through a novel CDE/CHR element in resting and G(1) cells. By relief of this repression in S and G(2) oscillating expression of cyclin B2 mRNA is achieved during the cell cycle.
Authors:
C Lange-zu Dohna; M Brandeis; F Berr; J Mössner; K Engeland
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  FEBS letters     Volume:  484     ISSN:  0014-5793     ISO Abbreviation:  FEBS Lett.     Publication Date:  2000 Nov 
Date Detail:
Created Date:  2000-11-17     Completed Date:  2000-12-07     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  0155157     Medline TA:  FEBS Lett     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  77-81     Citation Subset:  IM    
Affiliation:
Medizinische Klinik II, Department of Internal Medicine, University of Leipzig, Leipzig, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Base Sequence
Cell Cycle / genetics
Cells, Cultured
Cyclin B / genetics*,  metabolism
Cyclin B2
DNA / analysis
Gene Silencing
Mice
Molecular Sequence Data
Promoter Regions, Genetic / genetics*
Sequence Homology, Nucleic Acid
Simian virus 40 / genetics
Tandem Repeat Sequences / genetics*
Transcription, Genetic
Chemical
Reg. No./Substance:
0/Ccnb2 protein, mouse; 0/Cyclin B; 0/Cyclin B2; 9007-49-2/DNA

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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