| A tale of three PKCs: epsilon emerges as a driver of pre-neoplastic phenotypes. | |
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MedLine Citation:
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PMID: 21270526 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Authors:
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Karen E Knudsen |
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Publication Detail:
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Type: Comment; News Date: 2011-02-01 |
Journal Detail:
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Title: Cell cycle (Georgetown, Tex.) Volume: 10 ISSN: 1551-4005 ISO Abbreviation: Cell Cycle Publication Date: 2011 Feb |
Date Detail:
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Created Date: 2011-02-08 Completed Date: 2011-07-04 Revised Date: 2013-03-25 |
Medline Journal Info:
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Nlm Unique ID: 101137841 Medline TA: Cell Cycle Country: United States |
Other Details:
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Languages: eng Pagination: 379 Citation Subset: IM |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Animals Apoptosis Male Mice Mice, Transgenic Phenotype Precancerous Conditions / enzymology*, pathology Prostate / enzymology, pathology Prostatic Neoplasms / enzymology*, pathology Protein Kinase C-alpha / genetics, metabolism Protein Kinase C-delta / genetics, metabolism Protein Kinase C-epsilon / genetics*, metabolism Proto-Oncogene Proteins c-akt / metabolism STAT3 Transcription Factor / metabolism |
| Grant Support | |
ID/Acronym/Agency:
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R01 CA099996/CA/NCI NIH HHS |
| Chemical | |
Reg. No./Substance:
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0/STAT3 Transcription Factor; EC 2.7.11.1/Proto-Oncogene Proteins c-akt; EC 2.7.11.13/Protein Kinase C-alpha; EC 2.7.11.13/Protein Kinase C-delta; EC 2.7.11.13/Protein Kinase C-epsilon |
| Comments/Corrections | |
Comment On:
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Cell Cycle. 2011 Jan 15;10(2):268-77
[PMID:
21224724
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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