| Menière's symptoms resulting from bilateral otosclerotic occlusion of the endolymphatic duct: an analysis of a causal relationship between otosclerosis and Menière's disease. | |
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MedLine Citation:
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PMID: 2321690 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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An association between otosclerosis and Menière's disease has been proposed on both a clinical and temporal bone histopathologic basis for well over three-quarters of a century. Controversy persists over a causal relationship between these two entities, however, and the underlying pathophysiologic mechanisms relating capsular otosclerosis with Menière's disease remain speculative. The first case of total, bilateral endolymphatic duct occlusion resulting from extensive capsular otosclerosis is presented in a woman manifesting Menière's symptoms. The severe endolymphatic hydrops resulting from otosclerotic endolymphatic duct occlusion is given as histopathologic proof of a causal relationship between these two entities. Analysis of 18 of our own temporal bone cases of extensive capsular otosclerosis without endolymphatic hydrops and review of the literature indicates the uniqueness of such a firm causal relationship in the present case. |
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Authors:
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D J Franklin; A Pollak; U Fisch |
Publication Detail:
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Type: Case Reports; Journal Article |
Journal Detail:
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Title: The American journal of otology Volume: 11 ISSN: 0192-9763 ISO Abbreviation: Am J Otol Publication Date: 1990 Mar |
Date Detail:
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Created Date: 1990-05-10 Completed Date: 1990-05-10 Revised Date: 2007-11-15 |
Medline Journal Info:
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Nlm Unique ID: 7909513 Medline TA: Am J Otol Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 135-40 Citation Subset: IM |
Affiliation:
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Department of Otolaryngology, Baylor College of Medicine, Houston, Texas. |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Adult Endolymphatic Duct / pathology* Female Humans Meniere Disease / etiology*, pathology Otosclerosis / complications*, pathology Temporal Bone / pathology* Vestibule, Labyrinth / pathology* |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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