Document Detail


A sequel to the tale of p25/Cdk5 in neurodegeneration.
MedLine Citation:
PMID:  19081365     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
p25/Cdk5 dysregulation may contribute to neurodegeneration. In this issue of Neuron, Kim et al. show that cdk5 inactivates HDAC-1, leading to cell cycle deregulation and DNA damage accumulation. This study provides further insights into the function of p25/Cdk5 in neurons and points to HDAC-1 as a target for therapeutic interventions.
Authors:
Burcin Ikiz; Serge Przedborski
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Publication Detail:
Type:  Comment; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Review    
Journal Detail:
Title:  Neuron     Volume:  60     ISSN:  1097-4199     ISO Abbreviation:  Neuron     Publication Date:  2008 Dec 
Date Detail:
Created Date:  2008-12-16     Completed Date:  2009-04-08     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  8809320     Medline TA:  Neuron     Country:  United States    
Other Details:
Languages:  eng     Pagination:  731-2     Citation Subset:  IM    
Affiliation:
Department of Neurology, Pathology, and Cell Biology and Center for Motor Neuron Biology and Disease, Columbia University, New York, NY 10032, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cyclin-Dependent Kinase 5 / metabolism*
Humans
Models, Biological
Nerve Degeneration / enzymology*
Grant Support
ID/Acronym/Agency:
5R01AG21617-05/AG/NIA NIH HHS; P01 NS11766/NS/NINDS NIH HHS; P50 NS38370-09/NS/NINDS NIH HHS; R21 NS062180/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
EC 2.7.11.22/Cyclin-Dependent Kinase 5
Comments/Corrections
Comment On:
Neuron. 2008 Dec 10;60(5):803-17   [PMID:  19081376 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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