Document Detail


The role of fibrinogen degradation products in the pathogenesis of the respiratory distress syndrome.
MedLine Citation:
PMID:  918859     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Pulmonary dysfunction in awake rabbits was induced by intravenous infusion of a highly purified human fibrin split product (fragment D). The dose of infused fragment D was chosen to achieve observed plasma concentrations of fibrin split products in hospitalized patients with severe burns or trauma (about 100mug of FSP/ml of blood). Four hours after infusion, the animals displayed a clinical and pathological pattern which closely resembled post-traumatic acute respiratory distress syndrome, including hypoxia, hypocarbia, thrombocytopenia, increased pulmonary capillary permeability to albumin, interstitial edema, hypertrophy of alveolar lining cells, and intra-alveolar hemorrhage. In vivo production of fibrin split products by infusion of thrombin with induction of secondary fibrinolysis produced similar pulmonary changes, although intravascular clots and platelet aggregates also were prominent. Infusion of human fibrinogen and human albumin at identical doses failed to induce pulmonary dysfuction. The results suggest that fibrin split products (fragment D) alone are toxic to the respiratory system and may contribute to the development of acute respiratory distress syndrome in severely traumatized or burned patients.
Authors:
A Luterman; D Manwaring; P W Curreri
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Surgery     Volume:  82     ISSN:  0039-6060     ISO Abbreviation:  Surgery     Publication Date:  1977 Nov 
Date Detail:
Created Date:  1977-12-29     Completed Date:  1977-12-29     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0417347     Medline TA:  Surgery     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  703-9     Citation Subset:  AIM; IM    
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MeSH Terms
Descriptor/Qualifier:
Animals
Anoxia / etiology
Blood Platelets
Burns / blood
Carbon Dioxide / blood
Female
Fibrin Fibrinogen Degradation Products / pharmacology,  physiology*
Hemorrhage / etiology
Humans
Lung / pathology,  physiopathology
Male
Oxygen / blood
Pulmonary Edema / etiology
Rabbits
Respiratory Distress Syndrome, Adult / blood,  complications,  etiology*,  pathology
Thrombin / pharmacology,  physiology
Chemical
Reg. No./Substance:
0/Fibrin Fibrinogen Degradation Products; 124-38-9/Carbon Dioxide; 7782-44-7/Oxygen; EC 3.4.21.5/Thrombin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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