| The role of claudin-based tight junctions in morphogenesis. | |
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MedLine Citation:
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PMID: 19538288 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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In morphogenetic events during developmental processes, the hydrostatic pressure caused by fluid accumulation is thought to be an important factor determining the shape of epithelial structures. Recent studies using loss of function of claudins have demonstrated that tight junctions, by their control of fluid accumulation, have crucial roles in morphogenesis. Interestingly, both the barrier and the channel functions of tight junctions appear to contribute to morphogenesis. |
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Authors:
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Mikio Furuse; Kazumasa Moriwaki |
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Publication Detail:
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Type: Journal Article; Research Support, Non-U.S. Gov't |
Journal Detail:
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Title: Annals of the New York Academy of Sciences Volume: 1165 ISSN: 1749-6632 ISO Abbreviation: Ann. N. Y. Acad. Sci. Publication Date: 2009 May |
Date Detail:
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Created Date: 2009-06-22 Completed Date: 2009-08-03 Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 7506858 Medline TA: Ann N Y Acad Sci Country: United States |
Other Details:
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Languages: eng Pagination: 58-61 Citation Subset: IM |
Affiliation:
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Division of Cell Biology and Vascular Biology, Kobe University Graduate School of Medicine, Kobe, Japan. furuse@med.kobe-u.ac.jp |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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Animals Cell Membrane Permeability Embryo, Mammalian / metabolism Embryo, Nonmammalian / metabolism Fluorescent Antibody Technique Membrane Proteins / genetics, metabolism* Mice Morphogenesis / physiology* Tight Junctions / metabolism* Zebrafish |
| Chemical | |
Reg. No./Substance:
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0/Membrane Proteins; 0/claudin 12 protein, mouse; 0/claudin 15; 0/claudin 4; 0/claudin 6 |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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