Document Detail


The role of the atria in congenital heart disease.
MedLine Citation:
PMID:  12371005     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The atria play an important role in adult congenital heart disease. Atrial function is often altered due to longstanding pressure or volume overload. Cardiac surgery inflicts lasting damage to the atria, which leads to loss of atrial compliance. Both the history of atrial overload and the atrial scarring form substrates for atrial tachycardias. There has been a growing interest in the interatrial septum in the past few years. There is evidence for a role of the persisting foramen ovale and atrial septal aneurysm as a causative or permissive factor in cerebral stroke. Catheter closure of the PFO may be an attractive option, especially for younger patients.
Authors:
Folkert Meijboom; Jolien Roos-Hesselink; Horst Sievert
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Cardiology clinics     Volume:  20     ISSN:  0733-8651     ISO Abbreviation:  Cardiol Clin     Publication Date:  2002 Aug 
Date Detail:
Created Date:  2002-10-09     Completed Date:  2003-02-04     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8300331     Medline TA:  Cardiol Clin     Country:  United States    
Other Details:
Languages:  eng     Pagination:  351-66, v     Citation Subset:  IM    
Affiliation:
Department of Cardiology, Thoraxcentre, Erasmus MC, University Medical Center Rotterdam, P.O. Box 2040, 3000 CA, Rotterdam, The Netherlands. Meijboom@alkg.azr.nl
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MeSH Terms
Descriptor/Qualifier:
Adult
Arrhythmias, Cardiac / physiopathology*,  therapy
Atrial Function / physiology*
Cardiovascular Physiological Phenomena
Heart Aneurysm / complications
Heart Atria / physiopathology*
Heart Defects, Congenital / physiopathology*
Heart Septal Defects, Atrial / complications*,  therapy
Hemodynamics / physiology*
Humans
Stroke / complications,  prevention & control

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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