Document Detail


The role of T regulatory cells in human sepsis.
MedLine Citation:
PMID:  19345068     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
It is well-known that septic shock undermines immune homeostasis by inducing an initial intense systemic inflammatory response that is rapidly followed by a negative feedback of anti-inflammatory process. This secondary immunoparalysis state is characterized by decreased phagocytic cells, T cells, natural killer cells and B cells function and proinflammatory cytokine release. This persistence of immunoparalysis increased the risk for fatal outcome. In recent studies it was found that following the onset of septic shock, a relative increase in T regulatory cells number and suppressive function appears and makes them an important participant in the inhibition of immune responsiveness during sepsis. Consequently, a question emerging from these findings concerns the degree to which the manipulation of T regulatory cells might improve the outcome of patients with sepsis. Preliminary studies in animal models suggest that more work is needed to understand the conditions under which such a therapy may be effective.
Authors:
Aharon Kessel; Ellen Bamberger; Muhamad Masalha; Elias Toubi
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Publication Detail:
Type:  Journal Article; Review     Date:  2009-04-03
Journal Detail:
Title:  Journal of autoimmunity     Volume:  32     ISSN:  1095-9157     ISO Abbreviation:  J. Autoimmun.     Publication Date:    2009 May-Jun
Date Detail:
Created Date:  2009-05-08     Completed Date:  2009-07-21     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8812164     Medline TA:  J Autoimmun     Country:  England    
Other Details:
Languages:  eng     Pagination:  211-5     Citation Subset:  IM    
Affiliation:
Division of Allergy and Clinical Immunology, Bnai-Zion Medical Center, The Bruce Rappaport Faculty of Medicine, Technion, Haifa, Israel.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigens, CD / immunology,  metabolism
Cytokines / immunology*,  metabolism
Forkhead Transcription Factors / immunology,  metabolism
Humans
Immune Tolerance / immunology
Sepsis / immunology*,  metabolism
Shock, Septic / immunology,  metabolism
T-Lymphocytes, Regulatory / immunology*,  metabolism
Chemical
Reg. No./Substance:
0/Antigens, CD; 0/Cytokines; 0/Forkhead Transcription Factors; 0/cytotoxic T-lymphocyte antigen 4

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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