Document Detail


The role of APP proteolytic processing in lipid metabolism.
MedLine Citation:
PMID:  22179528     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
Amyloid plaques in brains are one of the major pathological hallmarks of Alzheimer's disease (AD). These plaques are mainly formed by aggregated Aβ, generated by proteolytic cleavage of the amyloid precursor protein (APP). Therefore, APP processing and Aβ production have been one of the central scopes in AD research in the past. Now, accumulating evidence suggests that besides its pathological impact, APP and its cleavage products also contribute to physiological functions. Proteolytic cleavage of APP is tightly regulated, and several lipids such as cholesterol and sphingolipids have been shown to influence APP processing and Aβ generation. In turn, Aβ as well as other APP cleavage products plays an essential role in regulating lipid homeostasis arguing for complex regulatory cycles in which lipids control APP processing and vice versa. This balanced regulation is disrupted under pathological conditions such as in AD. This article will review the physiological function of APP and its proteolytic products, especially Aβ and AICD, in regulating lipid homeostasis and which lipid species modulate APP processing. Furthermore, we summarize the alterations in lipid metabolism observed in AD patients and AD mouse models.
Authors:
Marcus O W Grimm; Tatjana L Rothhaar; Tobias Hartmann
Related Documents :
20331478 - Assessing the immunopotency of toll-like receptor agonists in an in vitro tissue-engine...
16133988 - No indication for a defect in toll-like receptor signaling in patients with hyper-ige s...
19917698 - Tlr cross-talk specifically regulates cytokine production by b cells from chronic infla...
16807108 - Tlrs, nlrs and rlrs: a trinity of pathogen sensors that co-operate in innate immunity.
17124428 - Role of cytokines in allergic airway inflammation.
15833068 - Immunomodulatory effects of human recombinant granulocyte-macrophage colony-stimulating...
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-12-17
Journal Detail:
Title:  Experimental brain research. Experimentelle Hirnforschung. Experimentation cerebrale     Volume:  -     ISSN:  1432-1106     ISO Abbreviation:  -     Publication Date:  2011 Dec 
Date Detail:
Created Date:  2011-12-19     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0043312     Medline TA:  Exp Brain Res     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Affiliation:
Deutsches Institut für DemenzPrävention (DIDP), Saarland University, Kirrbergerstrasse, 66421, Homburg/Saar, Germany, marcus.grimm@uks.eu.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Cervico-ocular coordination during neck rotation is distorted in people with whiplash-associated dis...
Next Document:  Age-related decline of peripheral visual processing: the role of eye movements.