Document Detail


A ribosomal protein L23-nucleophosmin circuit coordinates Mizl function with cell growth.
MedLine Citation:
PMID:  19160485     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The Myc-associated zinc-finger protein, Miz1, is a negative regulator of cell proliferation and induces expression of the cell-cycle inhibitors p15(Ink4b) and p21(Cip1). Here we identify the ribosomal protein L23 as a negative regulator of Miz1-dependent transactivation. L23 exerts this function by retaining nucleophosmin, an essential co-activator of Miz1 required for Miz1-induced cell-cycle arrest, in the nucleolus. Mutant forms of nucleophosmin found in acute myeloid leukaemia fail to co-activate Miz1 and re-localize it to the cytosol. As L23 is encoded by a direct target gene of Myc, this regulatory circuit may provide a feedback mechanism that links translation of Myc target genes and cell growth to Miz1-dependent cell-cycle arrest.
Authors:
Michael Wanzel; Annika C Russ; Daniela Kleine-Kohlbrecher; Emanuela Colombo; Pier-Guiseppe Pelicci; Martin Eilers
Related Documents :
17827275 - High-resolution timing of cell cycle-regulated gene expression.
10995475 - Combinatorial roles for prb, p107, and p130 in e2f-mediated cell cycle control.
2167175 - The identification of a second cell cycle control on the ho promoter in yeast: cell cyc...
3135985 - Light regulation of the cell cycle in euglena gracilis bacillaris.
15310925 - Positive selection.
6862175 - Immunocytochemical identification of the prolactin-secreting cells in the teleost pitui...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Nature cell biology     Volume:  10     ISSN:  1465-7392     ISO Abbreviation:  Nat. Cell Biol.     Publication Date:  2008 Sep 
Date Detail:
Created Date:  2009-01-22     Completed Date:  2009-02-04     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  100890575     Medline TA:  Nat Cell Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1051-61     Citation Subset:  IM    
Affiliation:
Institute for Molecular Biology and Tumor Research (IMT), Emil-Mannkopff-Str.2, 35033 Marburg, Germany.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Alleles
Animals
Cell Proliferation
Feedback, Physiological
Hela Cells
Humans
Models, Biological
Mutant Proteins / metabolism
Nuclear Proteins / metabolism*
Protein Inhibitors of Activated STAT / antagonists & inhibitors,  chemistry,  metabolism*
Protein Structure, Tertiary
Proto-Oncogene Proteins c-myc / metabolism
Rats
Ribosomal Proteins / metabolism*
Chemical
Reg. No./Substance:
0/Mutant Proteins; 0/Nuclear Proteins; 0/PIAS2 protein, human; 0/Protein Inhibitors of Activated STAT; 0/Proto-Oncogene Proteins c-myc; 0/Ribosomal Proteins; 0/ribosomal protein L17; 117896-08-9/nucleophosmin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Actin and alpha-actinin orchestrate the assembly and maturation of nascent adhesions in a myosin II ...
Next Document:  Talin depletion reveals independence of initial cell spreading from integrin activation and traction...