Document Detail


The retinoblastoma protein, RB, is required for gastrointestinal endocrine cells to exit the cell cycle, but not for hormone expression.
MedLine Citation:
PMID:  17936268     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Important functions of the RB family proteins include inhibition of cell cycle progression and regulation of terminal differentiation. We have examined the role of RB and the related protein, p107, in regulating cell cycle activity and differentiation of gastrointestinal endocrine cells, a relatively quiescent cell population, by conditionally disrupting the RB gene in neurogenin3 (Ngn3)-expressing cells in both p107(+/+) and p107(-/-) mice. Endocrine cells in the small intestine, colon, pancreas, and stomach were present in normal numbers in RB and RB-p107 mutants except for an increase in serotonin cells and decrease in ghrelin cells in the antral stomach. Deletion of RB resulted in a dramatic increase in proliferating serotonin cells in the antral stomach and intestine, whereas other enteroendocrine cell types exhibited much lower cell cycle activity or remained quiescent. The related p107 protein appears dispensable for enteroendocrine differentiation and does not functionally compensate for the loss of RB. Our results suggest that RB is required for enteroendocrine cells, particularly serotonin cells, to undergo cell cycle arrest as they terminally differentiate. RB has relatively subtle effects on enteroendocrine cell differentiation and is not required for the expression of the normal repertoire of hormones in the gastrointestinal tract.
Authors:
Yang Wang; Subir K Ray; Philip W Hinds; Andrew B Leiter
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2007-09-07
Journal Detail:
Title:  Developmental biology     Volume:  311     ISSN:  1095-564X     ISO Abbreviation:  Dev. Biol.     Publication Date:  2007 Nov 
Date Detail:
Created Date:  2007-11-12     Completed Date:  2008-03-24     Revised Date:  2010-12-03    
Medline Journal Info:
Nlm Unique ID:  0372762     Medline TA:  Dev Biol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  478-86     Citation Subset:  IM    
Affiliation:
Division of Gastroenterology, GRASP Digestive Disease Center, Tufts-New England Medical Center, Boston, MA 02111, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Basic Helix-Loop-Helix Transcription Factors / genetics,  metabolism
Cell Cycle / physiology*
Cell Differentiation / physiology*
Cells, Cultured
Enteroendocrine Cells / cytology,  metabolism*
Gastrointestinal Hormones / metabolism*
Gastrointestinal Tract / cytology*
Ghrelin / metabolism
Humans
Mice
Mice, Knockout
Nerve Tissue Proteins / genetics,  metabolism
Retinoblastoma Protein / genetics,  metabolism*
Retinoblastoma-Like Protein p107 / genetics,  metabolism
Serotonin / metabolism
Grant Support
ID/Acronym/Agency:
AG20208/AG/NIA NIH HHS; DK43673/DK/NIDDK NIH HHS; DK52870/DK/NIDDK NIH HHS; DK67166/DK/NIDDK NIH HHS; P30 DK034928-229002/DK/NIDDK NIH HHS; P30DK34928/DK/NIDDK NIH HHS; R01 AG020208-06/AG/NIA NIH HHS; R01 DK043673-14/DK/NIDDK NIH HHS; R01 DK043673-15/DK/NIDDK NIH HHS; R01 DK067166-03/DK/NIDDK NIH HHS; R01 DK067166-04/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Basic Helix-Loop-Helix Transcription Factors; 0/Gastrointestinal Hormones; 0/Ghrelin; 0/Nerve Tissue Proteins; 0/Neurog3 protein, mouse; 0/Retinoblastoma Protein; 0/Retinoblastoma-Like Protein p107; 50-67-9/Serotonin
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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