Document Detail


The renal physiology of pendrin (SLC26A4) and its role in hypertension.
MedLine Citation:
PMID:  17120771     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
SLC26A4 (pendrin, PDS) is a Na+-independent, Cl-/HCO3-/OH- exchanger that is expressed in the apical regions of type B and non-A, non-B intercalated cells within the cortical collecting duct (CCD), the connecting tubule and the distal convoluted tubule where it mediates HCO3- secretion and Cl- absorption. SLC26A4 is upregulated with aldosterone analogues and with Cl- restriction. While under basal conditions no renal abnormalities are observed in mice and humans with genetic disruption of SLC26A4 (Pendred syndrome), differences become apparent under conditions wherein the transporter is stimulated. Following treatment with aldosterone analogues, e.g. deoxycorticosterone pivalate (DOCP), weight gain and hypertension are observed in Slc26a4+/+ but not in Slc26a4-/- mice. During dietary NaCl restriction, a model in which serum aldosterone is appropriately increased, urinary volume and urinary excretion of Cl- are greater in Slc26a4-/- than in wild-type mice which results in apparent vascular volume contraction in Slc26a4-/- mice. Moreover, during NaCl restriction or following DOCP treatment, Slc26a4-/- mice have a higher serum HCO3- than wild type mice from an impaired ability to excrete OH- equivalents. In conclusion, SLC26A4 regulates blood pressure and arterial pH, likely by participating in the renal regulation of net acid and Cl- excretion.
Authors:
Susan M Wall
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Novartis Foundation symposium     Volume:  273     ISSN:  1528-2511     ISO Abbreviation:  Novartis Found. Symp.     Publication Date:  2006  
Date Detail:
Created Date:  2006-11-22     Completed Date:  2007-02-12     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  9807767     Medline TA:  Novartis Found Symp     Country:  England    
Other Details:
Languages:  eng     Pagination:  231-9; discussion 239-43, 261-4     Citation Subset:  IM    
Affiliation:
Renal Division, Emory University School of Medicine, 1639 Pierce Drive, NE Atlanta, GA 30322, USA.
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MeSH Terms
Descriptor/Qualifier:
Absorption / drug effects
Acid-Base Equilibrium / drug effects
Aldosterone / analogs & derivatives
Animals
Anion Transport Proteins / metabolism*
Bicarbonates / metabolism
Blood Pressure / drug effects
Body Weight / drug effects
Chlorides / metabolism
Cloning, Molecular
Hypertension / metabolism*,  physiopathology
Kidney / physiology*
Mice
Sodium Chloride / pharmacology
Systole / drug effects
Up-Regulation / drug effects
Water-Electrolyte Balance / drug effects
Grant Support
ID/Acronym/Agency:
DK 52935/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Anion Transport Proteins; 0/Bicarbonates; 0/Chlorides; 0/Slc26a4 protein, mouse; 52-39-1/Aldosterone; 7647-14-5/Sodium Chloride

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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