Document Detail


The protease inhibitor, elafin, induces p53-dependent apoptosis in human melanoma cells.
MedLine Citation:
PMID:  20020498     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Expression of the protease inhibitor elafin is deregulated in several human cancers. However, functions of the protein in cancer are yet to be established. Here, we show that elafin elicits pro-apoptotic effects in melanoma cells but not in normal melanocytes. Elafin triggered the intrinsic apoptotic pathway as evidenced by the increased caspase 9 activity and unaltered caspase 8 activity. Caspase 9-specific siRNA, but not caspase 8-specific siRNA, dramatically abrogated elafin-induced apoptosis. Elevated level of p53 was observed, resulting in increased transcriptional activation and consequent expression of downstream effector molecules (Bax, Puma, Noxa, p21). Moreover, the apoptotic effect of elafin was inhibited by p53-specific siRNA and the p53 inhibitor pifithrin-alpha. Elafin treatment of xenograft mice of melanoma cells led to significantly smaller tumor sizes compared with those of untreated control mice. Immunohistochemical analysis revealed decreased elafin expression in melanoma tissue specimens. Western blot and reverse transcription analyses indicated transcriptional repression of the elafin gene in melanoma cells. Our results collectively indicate that elafin induces apoptosis in melanoma cells through a p53-dependent intrinsic apoptotic pathway, and that repression of elafin expression in melanoma may contribute to disease progression.
Authors:
Kyung Sook Yu; Yangsoon Lee; Chung Mi Kim; Eui-Chul Park; Juhyun Choi; Dae-Sik Lim; Young-Hwa Chung; Sang Seok Koh
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International journal of cancer. Journal international du cancer     Volume:  127     ISSN:  1097-0215     ISO Abbreviation:  Int. J. Cancer     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-07-27     Completed Date:  2010-08-24     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0042124     Medline TA:  Int J Cancer     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1308-20     Citation Subset:  IM    
Affiliation:
Therapeutic Antibody Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon, Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects*,  physiology
Blotting, Western
Cell Line, Tumor
Cell Proliferation
Down-Regulation
Elafin / pharmacology*
Humans
Immunohistochemistry
Melanoma / metabolism,  pathology*
Mice
Mice, Nude
Neoplasm Transplantation
Protease Inhibitors / pharmacology*
RNA, Small Interfering
Recombinant Proteins / pharmacology
Reverse Transcriptase Polymerase Chain Reaction
Tumor Suppressor Protein p53 / physiology*
Chemical
Reg. No./Substance:
0/Elafin; 0/Protease Inhibitors; 0/RNA, Small Interfering; 0/Recombinant Proteins; 0/Tumor Suppressor Protein p53

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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