Document Detail


The pathophysiological mechanism underlying Brugada syndrome: depolarization versus repolarization.
MedLine Citation:
PMID:  20659475     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
This Point/Counterpoint presents a scholarly debate of the mechanisms underlying the electrocardiographic and arrhythmic manifestations of Brugada syndrome (BrS), exploring in detail the available evidence in support of the repolarization vs. depolarization hypothesis.
Authors:
Arthur A M Wilde; Pieter G Postema; José M Di Diego; Sami Viskin; Hiroshi Morita; Jeffrey M Fish; Charles Antzelevitch
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-07-24
Journal Detail:
Title:  Journal of molecular and cellular cardiology     Volume:  49     ISSN:  1095-8584     ISO Abbreviation:  J. Mol. Cell. Cardiol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-08-31     Completed Date:  2010-12-20     Revised Date:  2012-05-07    
Medline Journal Info:
Nlm Unique ID:  0262322     Medline TA:  J Mol Cell Cardiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  543-53     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Cardiology, Academic Medical Center Amsterdam, Amsterdam, The Netherlands.
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MeSH Terms
Descriptor/Qualifier:
Animals
Arrhythmias, Cardiac / physiopathology
Brugada Syndrome / physiopathology*
Cardiac Electrophysiology / methods
Electrocardiography
Humans
Grant Support
ID/Acronym/Agency:
HL47678/HL/NHLBI NIH HHS; R01 HL047678-16/HL/NHLBI NIH HHS; R01 HL047678-18/HL/NHLBI NIH HHS
Comments/Corrections

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