Document Detail


p53 is important for the anti-invasion of ganoderic acid T in human carcinoma cells.
MedLine Citation:
PMID:  21353507     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The function of p53 induced by ganoderic acids (GAs) in anti-invasion was unknown, although our previous work reported the inhibition of tumor invasion and metastasis by Ganoderic acid T (GA-T). This work indicated that GA-T promoted cell aggregation, inhibited cell adhesion and surpressed cell migration with a dose-dependent manner in human colon tumor cell lines of HCT-116 p53(+/+) and p53(-/-). Furthermore, comparing the ratios of HCT-116 p53(+/+) and p53(-/-) cells, p53 modified GA-T inhibition of migration and adhesion and GA-T promotion of cell aggregation, and p53 also modified GA-T inhibition of NF-κB nuclear translocation, IκBα degradation, and down-regulation of urokinase-type plaminogen activator (uPA), matrix metalloproteinase-2/9 (MMP-2/9), inducible nitric oxide synthase (iNOS/NOS2) protein expression and inducible nitric oxide (NO) production. The results indicated that p53 played an important role in anti-invasion of GA-T in human carcinoma cells. p53 may be an important target for GA-T inhibiting human carcinoma cells anti-invasion.
Authors:
Nian-Hong Chen; Jian-Jiang Zhong
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2011-02-25
Journal Detail:
Title:  Phytomedicine : international journal of phytotherapy and phytopharmacology     Volume:  18     ISSN:  1618-095X     ISO Abbreviation:  Phytomedicine     Publication Date:  2011 Jun 
Date Detail:
Created Date:  2011-06-07     Completed Date:  2011-10-14     Revised Date:  2011-10-27    
Medline Journal Info:
Nlm Unique ID:  9438794     Medline TA:  Phytomedicine     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  719-25     Citation Subset:  IM    
Copyright Information:
Copyright © 2011 Elsevier GmbH. All rights reserved.
Affiliation:
State Key Laboratory of Bioreactor Engineering, East China University of Science and Technology, Shanghai, PR China.
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MeSH Terms
Descriptor/Qualifier:
Antineoplastic Agents, Phytogenic / pharmacology*
Cell Adhesion / drug effects
Cell Aggregation / drug effects
Cell Line, Tumor
Cell Movement / drug effects
Colonic Neoplasms / drug therapy*,  metabolism,  pathology*
Drugs, Chinese Herbal / pharmacology
HCT116 Cells
Humans
I-kappa B Proteins / metabolism
Lanosterol / analogs & derivatives*,  pharmacology
Matrix Metalloproteinase 2 / biosynthesis
Matrix Metalloproteinase 9 / metabolism
Neoplasm Invasiveness
Nitric Oxide / metabolism
Nitric Oxide Synthase Type II / biosynthesis
Phytotherapy
Reishi / chemistry
Transcription Factor RelA / metabolism
Tumor Suppressor Protein p53 / metabolism*
Urokinase-Type Plasminogen Activator / biosynthesis
Chemical
Reg. No./Substance:
0/Antineoplastic Agents, Phytogenic; 0/Drugs, Chinese Herbal; 0/I-kappa B Proteins; 0/TP53 protein, human; 0/Transcription Factor RelA; 0/Tumor Suppressor Protein p53; 0/ganoderic acid T; 10102-43-9/Nitric Oxide; 139874-52-5/NF-kappaB inhibitor alpha; 79-63-0/Lanosterol; EC 1.14.13.39/NOS2 protein, human; EC 1.14.13.39/Nitric Oxide Synthase Type II; EC 3.4.21.73/Urokinase-Type Plasminogen Activator; EC 3.4.24.24/Matrix Metalloproteinase 2; EC 3.4.24.35/Matrix Metalloproteinase 9

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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