Document Detail


The overlap between neurodegenerative and vascular factors in the pathogenesis of dementia.
MedLine Citation:
PMID:  20623294     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
There is increasing evidence that cerebrovascular dysfunction plays a role not only in vascular causes of cognitive impairment but also in Alzheimer's disease (AD). Vascular risk factors and AD impair the structure and function of cerebral blood vessels and associated cells (neurovascular unit), effects mediated by vascular oxidative stress and inflammation. Injury to the neurovascular unit alters cerebral blood flow regulation, depletes vascular reserves, disrupts the blood-brain barrier, and reduces the brain's repair potential, effects that amplify the brain dysfunction and damage exerted by incident ischemia and coexisting neurodegeneration. Clinical-pathological studies support the notion that vascular lesions aggravate the deleterious effects of AD pathology by reducing the threshold for cognitive impairment and accelerating the pace of the dementia. In the absence of mechanism-based approaches to counteract cognitive dysfunction, targeting vascular risk factors and improving cerebrovascular health offers the opportunity to mitigate the impact of one of the most disabling human afflictions.
Authors:
Costantino Iadecola
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review     Date:  2010-07-11
Journal Detail:
Title:  Acta neuropathologica     Volume:  120     ISSN:  1432-0533     ISO Abbreviation:  Acta Neuropathol.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-07-28     Completed Date:  2010-11-15     Revised Date:  2011-09-13    
Medline Journal Info:
Nlm Unique ID:  0412041     Medline TA:  Acta Neuropathol     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  287-96     Citation Subset:  IM    
Affiliation:
Division of Neurobiology, Weill Cornell Medical College, New York, NY 10065, USA. coi2001@med.cornell.edu
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MeSH Terms
Descriptor/Qualifier:
Alzheimer Disease / pathology,  physiopathology
Animals
Brain / blood supply,  pathology*,  physiopathology
Cerebrovascular Circulation / physiology*
Cerebrovascular Disorders / pathology*,  physiopathology
Dementia / pathology*,  physiopathology
Humans
Nerve Degeneration / pathology*,  physiopathology
Grant Support
ID/Acronym/Agency:
HL96571/HL/NHLBI NIH HHS; NS37806/NS/NINDS NIH HHS; P01 HL096571-02/HL/NHLBI NIH HHS; R01 NS037853-13/NS/NINDS NIH HHS; R01 NS037853-14/NS/NINDS NIH HHS
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