Document Detail


A null allele frequent in non-Jewish Tay-Sachs patients.
MedLine Citation:
PMID:  8444467     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The molecular basis of null alleles was investigated by cDNA polymerase chain reaction (PCR) in seven Tay-Sachs patients. Although mRNAs were undetectable by Northern blot, cDNA-PCR amplification allowed us to get a sufficient amount of cDNA to characterize abnormal transcripts. In two French patients (one homozygote and one compound heterozygote with a 4-bp insertion in exon 11 of the second allele) suffering an infantile form of the disease, we detected abnormal RNAs with a 17-bp insertion due to a GT to AT transition at the donor site of intron 9, resulting in the activation of a cryptic donor site in the intron. This mutation has been found in 9 out of 82 Tay-Sachs chromosomes (11%) in association with alleles responsible from different clinical courses. In the other five patients we found the 4-bp insertion in exon 11 and two nonsense mutations.
Authors:
S Akli; J Chelly; A Kahn; L Poenaru
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Human genetics     Volume:  90     ISSN:  0340-6717     ISO Abbreviation:  Hum. Genet.     Publication Date:  1993 Feb 
Date Detail:
Created Date:  1993-04-05     Completed Date:  1993-04-05     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7613873     Medline TA:  Hum Genet     Country:  GERMANY    
Other Details:
Languages:  eng     Pagination:  614-20     Citation Subset:  IM    
Affiliation:
Laboratoire de Recherches en Génétique et Pathologie Moléculaires, INSERM, U. 129, CHU Cochin, Paris, France.
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MeSH Terms
Descriptor/Qualifier:
Alleles*
Base Sequence
Blotting, Northern
DNA Mutational Analysis
Exons
Female
Frameshift Mutation
Humans
Infant
Introns
Male
Molecular Sequence Data
Pedigree
Point Mutation
Polymerase Chain Reaction
RNA Splicing
RNA, Messenger / analysis
Tay-Sachs Disease / genetics*
Chemical
Reg. No./Substance:
0/RNA, Messenger

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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