Document Detail


A novel mechanism for neutrophil priming in trauma: potential role of peritoneal fluid.
MedLine Citation:
PMID:  20466401     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: We sought to determine the effect of peritoneal fluid from a novel animal model of abdominal compartment syndrome (ACS) on the proinflammatory status of polymorphonuclear leukocytes (PMNs) and monocytes. We hypothesize that peritoneal fluid is a potential priming and/or activating agent for PMNs/monocytes.
METHODS: ACS was induced in female Yorkshire swine, and peritoneal fluid was collected at the time of decompressive laparotomy. Naïve PMNs/monocytes were primed and/or activated with peritoneal fluid, phosphatidylcholine (PAF) plus peritoneal fluid, peritoneal fluid plus n-formyl-met-leu-phe (fMLP), and peritoneal fluid plus phorbol 12-myristate 13-acetate (PMA). Activation was determined by surface marker expression of integrins (CD11b an CD18) and selectins (CD62L). Additionally, proinflammatory cytokines in peritoneal fluid were analyzed.
RESULTS: Peritoneal fluid did not activate PMNs but increased CD11b expression on monocytes. When used as a primer for fMLP- or PMA-induced activation, peritoneal fluid significantly increased CD11b and CD18 expression on PMNs and monocytes. Peritoneal fluid collected at 6 and 12 h post decompressive laparotomy had similar effects. Interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) levels were increased in peritoneal fluid.
CONCLUSION: Peritoneal fluid represents a primer for PMNs/monocytes and seems to act through receptor-dependent and receptor-independent pathways. Strategies to reduce the amount of peritoneal fluid may decrease the locoregional and systemic inflammatory response by reducing priming and activation of neutrophils/monocytes.
Authors:
Shinil K Shah; Fernando Jimenez; Peter A Walker; Kevin R Aroom; Hasen Xue; Teri D Feeley; Karen S Uray; Kenneth C Norbury; Randolph H Stewart; Glen A Laine; Charles S Cox
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-05-13
Journal Detail:
Title:  Surgery     Volume:  148     ISSN:  1532-7361     ISO Abbreviation:  Surgery     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-16     Completed Date:  2010-08-06     Revised Date:  2011-09-26    
Medline Journal Info:
Nlm Unique ID:  0417347     Medline TA:  Surgery     Country:  United States    
Other Details:
Languages:  eng     Pagination:  263-70     Citation Subset:  AIM; IM    
Copyright Information:
Copyright 2010 Mosby, Inc. All rights reserved.
Affiliation:
Department of Pediatric Surgery, University of Texas Medical School at Houston, Houston, TX 77030, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Ascitic Fluid / physiology*
Compartment Syndromes / etiology,  physiopathology*,  surgery
Cytokines / physiology
Decompression, Surgical
Disease Models, Animal
Female
Inflammation Mediators / physiology
Interleukin-6 / physiology
Models, Biological
Monocytes / physiology
N-Formylmethionine Leucyl-Phenylalanine / pharmacology
Neutrophil Activation / drug effects,  physiology
Neutrophils / drug effects,  physiology*
Resuscitation / adverse effects
Sus scrofa
Tumor Necrosis Factor-alpha / physiology
Wounds and Injuries / complications,  physiopathology*
Grant Support
ID/Acronym/Agency:
K01 DK 070758/DK/NIDDK NIH HHS; K01 DK070758-04/DK/NIDDK NIH HHS; P50 GM 38529/GM/NIGMS NIH HHS; P50 GM038529-19/GM/NIGMS NIH HHS; R01 HL 36115/HL/NHLBI NIH HHS; R01 HL036115/HL/NHLBI NIH HHS; T32 GM 0879201/GM/NIGMS NIH HHS; T32 GM008792-09/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Cytokines; 0/Inflammation Mediators; 0/Interleukin-6; 0/Tumor Necrosis Factor-alpha; 59880-97-6/N-Formylmethionine Leucyl-Phenylalanine
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