Document Detail


A novel GJA1 missense mutation in a Polish child with oculodentodigital dysplasia.
MedLine Citation:
PMID:  19638688     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Oculodentodigital dysplasia (ODDD) (OMIM #164200) is a rare congenital, autosomal dominant disorder comprising craniofacial, ocular, dental, and digital anomalies. The syndrome is caused by GJA1 mutations. The clinical phenotype of ODDD involves a characteristic dysmorphic facies, ocular findings (microphthalmia, microcornea, glaucoma), syndactyly type III of the hands, phalangeal abnormalities, diffuse skeletal dysplasia, enamel dysplasia, and hypotrichosis. In a Polish child with the clinical symptoms typical of ODDD, we demonstrated a novel missense mutation c.C31A resulting in p.L11F substitution. Our report provides evidence on the importance of this highly conserved amino acid residue for the proper functioning of GJA1 protein.
Authors:
A Jamsheer; M Wisniewska; A Szpak; G Bugaj; M R Krawczynski; B Budny; A Wawrocka; A Latos-Bieleńska
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Publication Detail:
Type:  Case Reports; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of applied genetics     Volume:  50     ISSN:  1234-1983     ISO Abbreviation:  J. Appl. Genet.     Publication Date:  2009  
Date Detail:
Created Date:  2009-07-29     Completed Date:  2009-11-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9514582     Medline TA:  J Appl Genet     Country:  Poland    
Other Details:
Languages:  eng     Pagination:  297-9     Citation Subset:  IM    
Affiliation:
Chair and Department of Medical Genetics, Poland. jamsheer@wp.pl
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MeSH Terms
Descriptor/Qualifier:
Child, Preschool
Connexin 43 / genetics*
Eye Abnormalities / genetics*
Female
Humans
Mutation, Missense*
Syndactyly / genetics*
Syndrome
Tooth Abnormalities / genetics*
Chemical
Reg. No./Substance:
0/Connexin 43; 0/GJA1 protein, human

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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