Document Detail


A new ECG biomarker for drug toxicity: a combined signal processing and computational modeling study.
MedLine Citation:
PMID:  21096447     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
QT prolongation is the only clinically proven, yet insufficient, electrocardiogram (ECG) biomarker for drug-induced cardiac toxicity. The goal of this study is to evaluate whether JT area, i.e., total area of the T-wave, can serve as an ECG biomarker for drug-induced cardiac toxicity using both signal processing and computational modeling approaches. An ECG dataset that contained recordings from patients under control and sotalol condition was analyzed. In order to relate sotalol-induced ECG changes to its effect on ion channel level, i.e., blockade of the rapid component of the delayed rectifier potassium channel (I(Kr)), varied degrees of I(Kr) blockade were simulated in a slab of ventricular tissue. The mean JT area increased by 36.5% following the administration of sotalol in patients. Simulations in the slab tissue showed that sotalol increased action potential duration preferentially in the midmyocardium, which led to increased transmural dispersion of repolarization and JT area. In conclusion, JT area reflects the transmural dispersion of repolarization and may be a potentially useful surrogate/supplemental ECG biomarker to assess drug safety.
Authors:
Xiao Jie; Blanca Rodriguez; Esther Pueyo
Related Documents :
9294987 - Comparison of class ia/ib versus class iii antiarrhythmic drugs for the suppression of ...
11858397 - Torsade de pointes induced by metoclopramide in an elderly woman with preexisting compl...
19909327 - Arrhythmia, heart rate variability, and antiepileptic drugs.
596977 - A rapid in vivo technique for preliminary screening of antiarrhythmic agents in mice.
10810757 - Kinetics of a model nucleoside (guanosine) release from biodegradable poly(dl-lactide-c...
1100047 - Murine and human leukemias.
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Conference proceedings : ... Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Conference     Volume:  2010     ISSN:  1557-170X     ISO Abbreviation:  Conf Proc IEEE Eng Med Biol Soc     Publication Date:  2010  
Date Detail:
Created Date:  2010-11-24     Completed Date:  2011-03-30     Revised Date:  2011-08-01    
Medline Journal Info:
Nlm Unique ID:  101243413     Medline TA:  Conf Proc IEEE Eng Med Biol Soc     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2565-8     Citation Subset:  IM    
Affiliation:
Computing Laboratory, Oxford University, OX1 3QD, UK. xiao.jie@comlab.ox.ac.uk
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Action Potentials
Algorithms
Anti-Arrhythmia Agents / pharmacology,  toxicity*
Biological Markers
Computer Simulation
Electrocardiography / methods*
Humans
Ion Channels / chemistry
Ions
Models, Statistical
Signal Processing, Computer-Assisted
Software
Sotalol / pharmacology
Time Factors
Grant Support
ID/Acronym/Agency:
U24 HL096556-03/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Anti-Arrhythmia Agents; 0/Biological Markers; 0/Ion Channels; 0/Ions; 3930-20-9/Sotalol
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Analysis of QRS loop in the Vectorcardiogram of patients with Chagas' disease.
Next Document:  Using PIV to determine relative pressures in a stenotic phantom under steady flow based on the Press...