Document Detail


The neuronal expression of MYC causes a neurodegenerative phenotype in a novel transgenic mouse.
MedLine Citation:
PMID:  19164506     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Many different proteins associated with the cell cycle, including cyclins, cyclin-dependent kinases, and proto-oncogenes such as c-MYC (MYC), are increased in degenerating neurons. Consequently, an ectopic activation of the cell cycle machinery in neurons has emerged as a potential pathogenic mechanism of neuronal dysfunction and death in many neurodegenerative diseases, including Alzheimer's disease. However, the exact role of cell cycle re-entry during disease pathogenesis is unclear, primarily because of the lack of relevant research models to study the effects of cell cycle re-entry on mature neurons in vivo. To address this issue, we developed a new transgenic mouse model in which forebrain neurons (CaMKII-MYC) can be induced to enter the cell cycle using the physiologically relevant proto-oncogene MYC to drive cell cycle re-entry. We show that such cell cycle re-entry results in neuronal cell death, gliosis, and cognitive deficits. These findings provide compelling evidence that dysregulation of cell cycle re-entry results in neurodegeneration in vivo. Our current findings, coupled with those of previous reports, strengthen the hypothesis that neurodegeneration in Alzheimer's disease, similar to cellular proliferation in cancer, is a disease that results from inappropriate cell cycle control.
Authors:
Hyoung-gon Lee; Gemma Casadesus; Akihiko Nunomura; Xiongwei Zhu; Rudy J Castellani; Sandy L Richardson; George Perry; Dean W Felsher; Robert B Petersen; Mark A Smith
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2009-01-22
Journal Detail:
Title:  The American journal of pathology     Volume:  174     ISSN:  1525-2191     ISO Abbreviation:  Am. J. Pathol.     Publication Date:  2009 Mar 
Date Detail:
Created Date:  2009-02-23     Completed Date:  2009-03-23     Revised Date:  2013-01-31    
Medline Journal Info:
Nlm Unique ID:  0370502     Medline TA:  Am J Pathol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  891-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Pathology, Case Western Reserve University, 2103 Cornell Rd., Cleveland, OH 44106, USA. hyoung-gon.lee@case.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Cycle
Gene Expression Regulation
Genes, myc*
Heredodegenerative Disorders, Nervous System / genetics*,  pathology
Hippocampus / physiology
Humans
Maze Learning
Mice
Mice, Transgenic
Nerve Degeneration / genetics,  pathology
Neurons / physiology*
Phenotype
Reverse Transcriptase Polymerase Chain Reaction
Grant Support
ID/Acronym/Agency:
AG028679/AG/NIA NIH HHS; AG030096/AG/NIA NIH HHS; AG031364/AG/NIA NIH HHS; R21 AG030096/AG/NIA NIH HHS; R21 AG030096-02/AG/NIA NIH HHS
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