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A microdeletion at 12q24.31 can mimic beckwith-wiedemann syndrome neonatally.
MedLine Citation:
PMID:  20648245     Owner:  NLM     Status:  In-Data-Review    
Abstract/OtherAbstract:
We report on a patient who was initially suspected to have Beckwith-Wiedemann syndrome because of recurrent neonatal hypoglycaemias, macroglossia and overgrowth, but in whom no 11p15 abnormality could be found. Follow-up showed continued overgrowth and disturbed glucose homeostasis, a marked developmental delay, and severe behavioural problems especially caused by anxieties. Array comparative genomic hybridization analysis showed a de novo 12q24.31 interstitial deletion, which was confirmed by fluorescence in situ hybridization. The deleted region contains amongst others: HNF1 homeobox A (HNF1A) which is important for the regulation of gene expression in the liver and involved in maturity-onset diabetes of the young type 3 and insulin resistance; acyl-CoA dehydrogenase short chain (ACADS) which encodes an enzyme important in mitochondrial fatty acid beta-oxidation and can cause short-chain acyl-CoA dehydrogenese (SCAD) deficiency, and purinergic receptor P2X7 (P2RX7) which encodes a ligand-gated ion channel, and of which polymorphisms are found with increased frequency in patients with psychiatric disorders, especially anxieties. We conclude the present patient has a hitherto undescribed contiguous gene syndrome, which can initially resemble Beckwith-Wiedemann syndrome.
Authors:
E Baple; R Palmer; R C M Hennekam
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Publication Detail:
Type:  Journal Article     Date:  2010-01-11
Journal Detail:
Title:  Molecular syndromology     Volume:  1     ISSN:  1661-8777     ISO Abbreviation:  Mol Syndromol     Publication Date:  2010 Feb 
Date Detail:
Created Date:  2010-07-21     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101525192     Medline TA:  Mol Syndromol     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  42-5     Citation Subset:  -    
Affiliation:
Department of Clinical Genetics, Great Ormond Street Hospital for Children, UCL, London, UK.
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