Document Detail


microRNA miR-275 is indispensable for blood digestion and egg development in the mosquito Aedes aegypti.
MedLine Citation:
PMID:  21115818     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The mosquito Aedes aegypti is the major vector of arboviral diseases, particularly of Dengue fever, of which there are more than 100 million cases annually. Mosquitoes, such as A. aegypti, serve as vectors for disease pathogens because they require vertebrate blood for their egg production. Pathogen transmission is tightly linked to repeated cycles of obligatory blood feeding and egg maturation. Thus, the understanding of mechanisms governing egg production is necessary to develop approaches that limit the spread of mosquito-borne diseases. Previous studies have identified critical roles of hormonal- and nutrition-based target of rapamycin (TOR) pathways in controlling blood-meal-mediated egg maturation in mosquitoes. In this work, we uncovered another essential regulator of blood-meal-activated processes, the microRNA miR-275. The depletion of this microRNA in A. aegypti females after injection of its specific antagomir resulted in severe defects in blood digestion, fluid excretion, and egg development, clearly demonstrating that miR-275 is indispensable for these physiological processes. miR-275 exhibits an expression profile that suggests its regulation by a steroid hormone, 20-hydroxyecdysone (20E). In vitro organ culture experiments demonstrated that miR-275 is induced by this hormone in the presence of amino acids, indicative of a dual regulation by 20E and TOR. This report has uncovered the critical importance of microRNAs in controlling blood-meal-activated physiological events required for completion of egg development in mosquito disease vectors.
Authors:
Bart Bryant; Warren Macdonald; Alexander S Raikhel
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-11-29
Journal Detail:
Title:  Proceedings of the National Academy of Sciences of the United States of America     Volume:  107     ISSN:  1091-6490     ISO Abbreviation:  Proc. Natl. Acad. Sci. U.S.A.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-12-29     Completed Date:  2011-03-01     Revised Date:  2011-08-01    
Medline Journal Info:
Nlm Unique ID:  7505876     Medline TA:  Proc Natl Acad Sci U S A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  22391-8     Citation Subset:  IM    
Affiliation:
Department of Entomology, Institute for Integrative Genome Biology, University of California, Riverside, CA 92521, USA.
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MeSH Terms
Descriptor/Qualifier:
Aedes / genetics*,  metabolism,  physiology
Amino Acid Sequence
Animals
Blood / metabolism
Blotting, Western
Ecdysterone / pharmacology
Fat Body / metabolism*
Feeding Behavior
Female
Gene Expression Profiling
Gene Expression Regulation / drug effects
Insect Proteins / genetics,  metabolism
MicroRNAs / genetics*,  metabolism
Molecular Sequence Data
Oocytes / growth & development,  metabolism*
RNA Interference
Rats
Reverse Transcriptase Polymerase Chain Reaction
Sequence Homology, Amino Acid
Signal Transduction
TOR Serine-Threonine Kinases / genetics,  metabolism
Tissue Culture Techniques
Grant Support
ID/Acronym/Agency:
R37 AI024716-24/AI/NIAID NIH HHS; R37 AI244716/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Insect Proteins; 0/MicroRNAs; 0/microRNA miR-275, Aedes aegypti; 5289-74-7/Ecdysterone; EC 2.7.1.1/TOR Serine-Threonine Kinases
Comments/Corrections

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