Document Detail


The influence of MK-801 on bicuculline evoked seizures in adult mice exposed to transient episode of brain ischemia.
MedLine Citation:
PMID:  11041274     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The aim of the study was to examine the role of NMDA receptors in modulation of protective effect against bicuculline toxicity after transient brain ischemia in mice. Animals were exposed for 30 min to bilateral clamping of the common carotid arteries (BCCA) in anaesthesia. MK-801 was administered intraperitoneally in two paradigms: a) acute treatment: twice, 1.0 mg/kg; 1 hour before clamping and 6 hours after re-circulation and b) chronic treatment: 0.1 mg/kg; started 24 hours after re-circulation and continued once daily for 13 days, the last injection was administered 24 hours before seizure induction. 14 days after BCCA, the animals were injected with bicuculline (3.5 mg/kg s.c). A significant decrease in seizure susceptibility could be observed in BCCA treated mice compared with sham-operated controls. Acute treatment with MK-801 did not affect seizure activity both in sham and BCCA mice. Chronic treatment with the drug potentiated anticonvulsant effect of brain ischemia but had no influence on seizure activity in sham-operated mice. The analysis of GABA content in brain tissue performed 14 days after BCCA showed a moderate increase in vehicle-treated mice and significant elevation after chronic treatment with MK-801. It can be suggested that NMDA receptors are not involved in the induction of a protective effect against bicuculline toxicity after transient brain ischemia. The prolonged treatment with low doses of MK-801 may potentiate a developed process in a mechanism of chemical preconditioning.
Authors:
K Rejdak; R Rejdak; Z Kleinrok; M Sieklucka-Dziuba
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of neural transmission (Vienna, Austria : 1996)     Volume:  107     ISSN:  0300-9564     ISO Abbreviation:  J Neural Transm     Publication Date:  2000  
Date Detail:
Created Date:  2001-01-26     Completed Date:  2001-02-15     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  9702341     Medline TA:  J Neural Transm     Country:  AUSTRIA    
Other Details:
Languages:  eng     Pagination:  947-57     Citation Subset:  IM    
Affiliation:
Department of Pharmacology, Medical University, Lublin, Poland.
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MeSH Terms
Descriptor/Qualifier:
Age Factors
Animals
Behavior, Animal / drug effects
Bicuculline
Brain Chemistry / drug effects
Convulsants
Dizocilpine Maleate / pharmacology*
Epilepsy / chemically induced,  drug therapy*,  etiology
Excitatory Amino Acid Antagonists / pharmacology*
Ischemic Attack, Transient / complications,  drug therapy*,  mortality
Ischemic Preconditioning
Male
Mice
Receptors, N-Methyl-D-Aspartate / antagonists & inhibitors,  metabolism
Seizures / chemically induced,  drug therapy,  etiology
Survival Rate
gamma-Aminobutyric Acid / analysis,  metabolism
Chemical
Reg. No./Substance:
0/Convulsants; 0/Excitatory Amino Acid Antagonists; 0/Receptors, N-Methyl-D-Aspartate; 485-49-4/Bicuculline; 56-12-2/gamma-Aminobutyric Acid; 77086-22-7/Dizocilpine Maleate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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