Document Detail


The immunopathophysiology of multiple sclerosis.
MedLine Citation:
PMID:  21439440     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
This review explores the principle features of the immunopathology of multiple sclerosis (MS), particularly relapsing-remitting MS. It highlights the emerging concepts in the pathogenesis of MS in the context of known features of pathology, including the characterization of cytokine networks promoting inflammatory damage of the central nervous system, B-cell involvement, and inflammatory damage of axons and neurons. This article preferentially focuses on MS rather than animal models of the disease, such as experimental autoimmune encephalomyelitis.
Authors:
Gregory F Wu; Enrique Alvarez
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Neurologic clinics     Volume:  29     ISSN:  1557-9875     ISO Abbreviation:  Neurol Clin     Publication Date:  2011 May 
Date Detail:
Created Date:  2011-03-28     Completed Date:  2011-07-19     Revised Date:  2012-05-02    
Medline Journal Info:
Nlm Unique ID:  8219232     Medline TA:  Neurol Clin     Country:  United States    
Other Details:
Languages:  eng     Pagination:  257-78     Citation Subset:  IM    
Copyright Information:
Copyright © 2011 Elsevier Inc. All rights reserved.
Affiliation:
Division of Multiple Sclerosis, Department of Neurology, Washington University, Box 8111, 660 South Euclid Avenue, St Louis, MO 63110, USA. wug@neuro.wustl.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Antigen-Presenting Cells / pathology
Axons / pathology
Brain / pathology
Cytokines / metabolism
Humans
Leukocytes / physiology
Lymphocytes / physiology
Models, Biological
Multiple Sclerosis / immunology*,  metabolism,  pathology*
Neurons / pathology
Grant Support
ID/Acronym/Agency:
K08 NS062138-04/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Cytokines

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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