Document Detail


A human gene responsible for Zellweger syndrome that affects peroxisome assembly.
MedLine Citation:
PMID:  1546315     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The primary defect arising from Zellweger syndrome appears to be linked to impaired assembly of peroxisomes. A human complementary DNA has been cloned that complements the disease's symptoms (including defective peroxisome assembly) in fibroblasts from a patient with Zellweger syndrome. The cause of the syndrome in this patient was a point mutation that resulted in the premature termination of peroxisome assembly factor-1. The homozygous patient apparently inherited the mutation from her parents, each of whom was heterozygous for that mutation.
Authors:
N Shimozawa; T Tsukamoto; Y Suzuki; T Orii; Y Shirayoshi; T Mori; Y Fujiki
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Publication Detail:
Type:  Case Reports; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Science (New York, N.Y.)     Volume:  255     ISSN:  0036-8075     ISO Abbreviation:  Science     Publication Date:  1992 Feb 
Date Detail:
Created Date:  1992-04-15     Completed Date:  1992-04-15     Revised Date:  2007-03-19    
Medline Journal Info:
Nlm Unique ID:  0404511     Medline TA:  Science     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1132-4     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Gifu University School of Medicine, Japan.
Data Bank Information
Bank Name/Acc. No.:
GENBANK/M86852
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Sequence
Animals
Base Sequence
Cricetinae
DNA Mutational Analysis
Genes
Genetic Complementation Test
Humans
Membrane Proteins / genetics*
Microbodies / ultrastructure*
Molecular Sequence Data
Oligodeoxyribonucleotides / chemistry
Pedigree
Polymerase Chain Reaction
Transfection
Zellweger Syndrome / genetics*
Chemical
Reg. No./Substance:
0/Membrane Proteins; 0/Oligodeoxyribonucleotides; 135847-86-8/peroxisome assembly factor-1

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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