Document Detail


hnRNP G elicits tumor-suppressive activity in part by upregulating the expression of Txnip.
MedLine Citation:
PMID:  18541147     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Heterogeneous nuclear ribonuclearproteins (hnRNPs) are nucleic acid-binding proteins and have critical roles in DNA repair, telomere regulation, and transcriptional gene regulation. Previously, we showed that hnRNP G has tumor-suppressive activity in human oral squamous cell carcinoma cells. Therefore, the identification of hnRNP G target genes is important for understanding the function of hnRNP G and its tumor-suppressive activity. In this study, we identify a known tumor suppressor gene, thioredoxin-interacting protein (Txnip) gene as a novel target of hnRNP G. Expression of Txnip is upregulated by wild-type (wt) hnRNP G but not by a suppression-defective mutant hnRNP G (K22R) in human squamous cell carcinoma. Wt hnRNP G binds and transactivates the Txnip promoter in vivo, whereas the K22R mutant does not. Furthermore, overexpression of Txnip alone in cancer cells leads to the inhibition of anchorage-independent growth and in vivo tumorigenicity in immunocompromised mice, suggesting a reversion of the transformation phenotype. These studies indicate that hnRNP G promotes the expression of Txnip and mediates its tumor-suppressive effect.
Authors:
Ki-Hyuk Shin; Reuben H Kim; Roy H Kim; Mo K Kang; No-Hee Park
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2008-06-09
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  372     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2008 Aug 
Date Detail:
Created Date:  2008-06-25     Completed Date:  2008-07-24     Revised Date:  2009-11-18    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  880-5     Citation Subset:  IM    
Affiliation:
School of Dentistry, University of California, CHS 43-033, 10833 Le Conte Avenue, Los Angeles, CA 90095, USA. kshin@dentistry.ucla.edu
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MeSH Terms
Descriptor/Qualifier:
Carrier Proteins / genetics*
Cell Line, Tumor
Cell Proliferation
Gene Expression Regulation, Neoplastic*
Heterogeneous-Nuclear Ribonucleoproteins / genetics,  metabolism*
Humans
Promoter Regions, Genetic
Transcriptional Activation*
Tumor Suppressor Proteins / genetics,  metabolism*
Up-Regulation
Grant Support
ID/Acronym/Agency:
DE14147/DE/NIDCR NIH HHS; DE15902/DE/NIDCR NIH HHS; R03 DE015902-01/DE/NIDCR NIH HHS; R03 DE015902-02/DE/NIDCR NIH HHS
Chemical
Reg. No./Substance:
0/Carrier Proteins; 0/Heterogeneous-Nuclear Ribonucleoproteins; 0/TXNIP protein, human; 0/Tumor Suppressor Proteins; 0/heterogeneous nuclear ribonucleoprotein G, human
Comments/Corrections

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