Document Detail


The heterogeneous response of the bivascularly perfused rat liver to adenosine.
MedLine Citation:
PMID:  10424758     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The heterogeneity of the liver parenchyma in relation to the metabolic response to adenosine was investigated using the bivascularly perfused rat liver in the anterograde and retrograde modes. Adenosine was infused into livers from fed rats according to four experimental protocols: (A) anterograde perfusion, adenosine via the portal vein; (B) anterograde perfusion, adenosine via the hepatic artery; (C) retrograde perfusion, adenosine via the hepatic vein; and (D) retrograde perfusion, adenosine via the hepatic artery. Due to the very pronounced concentration gradients generated by metabolic transformation, the infused adenosine attained maximal concentrations in different regions with each experimental protocol. The sinusoidal mean transit times (t(s)) were not changed by adenosine in anterograde perfusion, but were increased in retrograde perfusion. It was concluded that the vasoconstrictive elements are localized essentially in the presinusoidal region. Glucose release stimulation presented two kinetic components. The first one was rapid in both onset and decay with a peak around 30 sec; the second one developed more slowly (several minutes). The factors of the first kinetic component are possibly generated in the presinusoidal region or in the first periportal cells. The initial decrease in oxygen consumption seemed to be localized in the region just after the intrasinusoidal confluence of the ramifications of the portal vein and hepatic artery. Indomethacin decreased glucose release stimulation by adenosine in both anterograde and retrograde perfusion only when DMSO was the vehicle. The participation of eicosanoids in the generation of the effects of adenosine seems to be less important than hitherto believed.
Authors:
T R Fernandes; F Suzuki-Kemmelmeier; E C Prado; A Bracht
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Biochemical pharmacology     Volume:  58     ISSN:  0006-2952     ISO Abbreviation:  Biochem. Pharmacol.     Publication Date:  1999 Aug 
Date Detail:
Created Date:  1999-08-02     Completed Date:  1999-08-02     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0101032     Medline TA:  Biochem Pharmacol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  397-409     Citation Subset:  IM    
Affiliation:
Laboratory of Liver Metabolism, University of Maringa, Brazil.
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MeSH Terms
Descriptor/Qualifier:
Adenosine / metabolism,  pharmacology*
Animals
Drug Interactions
Eicosanoids / pharmacology
Glucose / metabolism
Hemodynamics / drug effects
Indomethacin / pharmacology
Kinetics
Liver / drug effects*,  physiology
Male
Oxygen / metabolism
Perfusion
Rats
Rats, Wistar
Vasodilator Agents / metabolism,  pharmacology*
Chemical
Reg. No./Substance:
0/Eicosanoids; 0/Vasodilator Agents; 50-99-7/Glucose; 53-86-1/Indomethacin; 58-61-7/Adenosine; 7782-44-7/Oxygen

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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