Document Detail


The hedgehog receptor patched is involved in cholesterol transport.
MedLine Citation:
PMID:  21931618     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Sonic hedgehog (Shh) signaling plays a crucial role in growth and patterning during embryonic development, and also in stem cell maintenance and tissue regeneration in adults. Aberrant Shh pathway activation is involved in the development of many tumors, and one of the most affected Shh signaling steps found in these tumors is the regulation of the signaling receptor Smoothened by the Shh receptor Patched. In the present work, we investigated Patched activity and the mechanism by which Patched inhibits Smoothened.
METHODOLOGY/PRINCIPAL FINDINGS: Using the well-known Shh-responding cell line of mouse fibroblasts NIH 3T3, we first observed that enhancement of the intracellular cholesterol concentration induces Smoothened enrichment in the plasma membrane, which is a crucial step for the signaling activation. We found that binding of Shh protein to its receptor Patched, which involves Patched internalization, increases the intracellular concentration of cholesterol and decreases the efflux of a fluorescent cholesterol derivative (BODIPY-cholesterol) from these cells. Treatment of fibroblasts with cyclopamine, an antagonist of Shh signaling, inhibits Patched expression and reduces BODIPY-cholesterol efflux, while treatment with the Shh pathway agonist SAG enhances Patched protein expression and BODIPY-cholesterol efflux. We also show that over-expression of human Patched in the yeast S. cerevisiae results in a significant boost of BODIPY-cholesterol efflux. Furthermore, we demonstrate that purified Patched binds to cholesterol, and that the interaction of Shh with Patched inhibits the binding of Patched to cholesterol.
CONCLUSION/SIGNIFICANCE: Our results suggest that Patched may contribute to cholesterol efflux from cells, and to modulation of the intracellular cholesterol concentration. This activity is likely responsible for the inhibition of the enrichment of Smoothened in the plasma membrane, which is an important step in Shh pathway activation.
Authors:
Michel Bidet; Olivier Joubert; Benoit Lacombe; Marine Ciantar; Rony Nehmé; Patrick Mollat; Lionel Brétillon; Hélène Faure; Robert Bittman; Martial Ruat; Isabelle Mus-Veteau
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2011-09-08
Journal Detail:
Title:  PloS one     Volume:  6     ISSN:  1932-6203     ISO Abbreviation:  PLoS ONE     Publication Date:  2011  
Date Detail:
Created Date:  2011-09-20     Completed Date:  2012-02-03     Revised Date:  2012-04-26    
Medline Journal Info:
Nlm Unique ID:  101285081     Medline TA:  PLoS One     Country:  United States    
Other Details:
Languages:  eng     Pagination:  e23834     Citation Subset:  IM    
Affiliation:
Université de Nice-Sophia Antipolis, CNRS-UMR 6543, Institute of Developmental Biology and Cancer, Nice, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Biological Transport / drug effects
Cell Membrane / drug effects,  metabolism
Cholesterol / metabolism*
Hedgehog Proteins / metabolism
Humans
Mice
NIH 3T3 Cells
Receptors, Cell Surface / genetics,  metabolism*
Receptors, G-Protein-Coupled / metabolism
Saccharomyces cerevisiae / genetics
Signal Transduction / drug effects
Veratrum Alkaloids / pharmacology
Chemical
Reg. No./Substance:
0/Hedgehog Proteins; 0/Receptors, Cell Surface; 0/Receptors, G-Protein-Coupled; 0/Smo protein, mouse; 0/Veratrum Alkaloids; 0/patched receptors; 4449-51-8/cyclopamine; 57-88-5/Cholesterol
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