Document Detail


A genetic screen for behavioral mutations that perturb dopaminergic homeostasis in mice.
MedLine Citation:
PMID:  16436185     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Disruption of dopaminergic (DA) systems is thought to play a central role in the addictive process and in the pathophysiology of schizophrenia. Although inheritance plays an important role in the predisposition to these disorders, the genetic basis of this is not well understood. To provide additional insight, we have performed a modifier screen in mice designed to identify mutations that perturb DA homeostasis. With a genetic background sensitized by a mutation in the dopamine transporter (DAT), we used random chemical mutagenesis and screened for mutant mice with locomotor abnormalities. Four mutant lines were identified with quantitatively elevated levels of locomotor activity. Mapping of mutations in these lines identified two loci that alter activity only when dopamine levels are elevated by a DAT mutation and thus would only have been uncovered by this type of approach. One of these quantitative trait loci behaves as an enhancer of DA neurotransmission, whereas the other may act as a suppressor. In addition, we also identified three loci which are not dependent on the sensitized background but which also contribute to the overall locomotor phenotype.
Authors:
D J Speca; N Rabbee; D Chihara; T P Speed; A S Peterson
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.    
Journal Detail:
Title:  Genes, brain, and behavior     Volume:  5     ISSN:  1601-1848     ISO Abbreviation:  Genes Brain Behav.     Publication Date:  2006 Feb 
Date Detail:
Created Date:  2006-01-26     Completed Date:  2006-05-03     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  101129617     Medline TA:  Genes Brain Behav     Country:  England    
Other Details:
Languages:  eng     Pagination:  19-28     Citation Subset:  IM    
Affiliation:
Department of Neurology and the Ernest Gallo Clinic and Research Center, University of California at San Francisco, Emeryville, CA 94608, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Chromosome Mapping
Dopamine / metabolism*
Dopamine Plasma Membrane Transport Proteins / genetics,  metabolism*
Ethylnitrosourea
Exploratory Behavior / physiology*
Female
Genetic Testing*
Heterozygote Detection
Homeostasis / genetics*
Homozygote
Male
Mice
Mice, Inbred DBA
Mice, Knockout
Motor Activity / genetics*
Mutagenesis / genetics*
Mutagens
Phenotype
Quantitative Trait Loci / genetics
Random Allocation
Chemical
Reg. No./Substance:
0/Dopamine Plasma Membrane Transport Proteins; 0/Mutagens; 759-73-9/Ethylnitrosourea

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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