Document Detail


The final fates of neurogenin2-expressing cells include all major neuron types in the mouse retina.
MedLine Citation:
PMID:  16364654     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The molecular mechanism underlying vertebrate retinal development is not well understood. To examine whether neurogenin2 (ngn2) expression determines cell fate in the retina, we mapped the final fates of cells that once expressed ngn2, using the conditional, binary CreER -LacZ system. We found LacZ+ cells in all 3 nuclear layers of the mouse retina and including all major types of neurons: photoreceptors, horizontal, bipolar, amacrine, and ganglion cells. The distribution of LacZ+ cells among the 3 nuclear layers closely resembled a theoretical distribution of total retinal cells. The temporal window in which each cell type was marked appeared nonrandom, and was similar to its birthdate and birth sequence. These data indicate that cells expressing ngn2 at some point in their life histories may later differentiate into a variety of cell types.
Authors:
Wenxin Ma; Shu-Zhen Wang
Related Documents :
11731454 - Notch-delta signaling induces a transition from mitotic cell cycle to endocycle in dros...
21636684 - Effect of nickel and chromium exposure on buccal cells of electroplaters.
9888854 - Xid-like immunodeficiency in mice with disruption of the p85alpha subunit of phosphoino...
22874794 - Fabrication and characteristics of zno/oad-inn/pbpc hybrid solar cells prepared by obli...
16900214 - Endothelial cell-driven regulation of cd9 or motility-related protein-1 expression in m...
17610204 - Phosphoproteomic analysis of neuronal cell death by glutamate-induced oxidative stress.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2005-12-20
Journal Detail:
Title:  Molecular and cellular neurosciences     Volume:  31     ISSN:  1044-7431     ISO Abbreviation:  Mol. Cell. Neurosci.     Publication Date:  2006 Mar 
Date Detail:
Created Date:  2006-03-13     Completed Date:  2006-05-22     Revised Date:  2012-05-02    
Medline Journal Info:
Nlm Unique ID:  9100095     Medline TA:  Mol Cell Neurosci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  463-9     Citation Subset:  IM    
Affiliation:
Department of Ophthalmology, University of Alabama at Birmingham, 700 South 18th Street, Birmingham, AL 35294-0009, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Amacrine Cells / cytology,  metabolism
Animals
Basic Helix-Loop-Helix Transcription Factors / biosynthesis*,  genetics
Cell Differentiation / physiology*
Cell Lineage / physiology*
Cell Proliferation
Female
Gene Expression Regulation, Developmental / physiology
Genes, Reporter / genetics
Lac Operon / genetics
Male
Mice
Mice, Transgenic
Nerve Tissue Proteins / biosynthesis*,  genetics
Neurons / cytology,  metabolism*
Photoreceptor Cells, Vertebrate / cytology,  metabolism
Retina / cytology,  embryology*,  metabolism
Retinal Bipolar Cells / cytology,  metabolism
Retinal Ganglion Cells / cytology,  metabolism
Stem Cells / cytology,  metabolism*
Grant Support
ID/Acronym/Agency:
EY11640/EY/NEI NIH HHS; R01 EY011640/EY/NEI NIH HHS; R01 EY011640-07/EY/NEI NIH HHS
Chemical
Reg. No./Substance:
0/Basic Helix-Loop-Helix Transcription Factors; 0/Nerve Tissue Proteins; 0/Neurog2 protein, mouse
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Clustering transmembrane-agrin induces filopodia-like processes on axons and dendrites.
Next Document:  Gi/o proteins: expression for direct activation enquiry.