Document Detail


The effects of endothelial factor inhibition on the time course of responses of isolated rat coronary arteries to intraluminal flow.
MedLine Citation:
PMID:  17337908     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The aims of this study were to investigate, for the first time, the effects of endothelial factor inhibition on both the magnitude and dynamics of the response of isolated small coronary arteries to intraluminal flow. Isolated rat coronary arteries were mounted on a pressure myograph and left to develop myogenic tone. Flow was introduced and maintained until stable diameters were attained. Dilatory responses were observed which were maximal at low flow rates (5-10 microl/min) and thus shear stresses (1-2 dyn/cm(2)). These responses were transient in nature. Transient dilations were also observed upon cessation of flow. All responses (to 5 microl/min) were endothelium dependent and were completely abolished by addition of charybdotoxin (100 nM) and apamin (100-500 nM) suggesting an important role for a hyperpolarizing mechanism most likely involving an endothelium-derived hyperpolarizing factor. However, inhibitors of nitric oxide synthase (L-NNA; 100 microM) or cyclo-oxygenase (indomethacin; 10 microM) also modulated the response causing an increase and decrease in maximum vasodilation, respectively. By examining the time course we showed that both agents also made the response significantly more transient in nature. These results show that inhibition of endothelial factor pathways can influence both the magnitude and dynamics of the response of isolated rat coronary arteries to flow.
Authors:
May Azzawi; Clare Austin
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Publication Detail:
Type:  In Vitro; Journal Article     Date:  2007-02-28
Journal Detail:
Title:  Journal of vascular research     Volume:  44     ISSN:  1018-1172     ISO Abbreviation:  J. Vasc. Res.     Publication Date:  2007  
Date Detail:
Created Date:  2007-04-13     Completed Date:  2007-05-24     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9206092     Medline TA:  J Vasc Res     Country:  Switzerland    
Other Details:
Languages:  eng     Pagination:  223-33     Citation Subset:  IM    
Affiliation:
Smooth Muscle Physiology Group, Division of Cardiovascular and Endocrine Sciences, Core Technology Facility, University of Manchester, Manchester, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apamin / pharmacology
Biological Factors / metabolism
Charybdotoxin / pharmacology
Coronary Vessels / drug effects,  metabolism*
Cyclooxygenase Inhibitors / pharmacology
Endothelium, Vascular / drug effects,  metabolism*
Indomethacin / pharmacology
Male
Myography
Nitric Oxide / metabolism
Nitric Oxide Synthase / antagonists & inhibitors
Nitroarginine / pharmacology
Potassium Channel Blockers / pharmacology
Pressure
Prostaglandins / metabolism
Pulsatile Flow
Rats
Rats, Wistar
Stress, Mechanical
Time Factors
Vasodilation* / drug effects
Chemical
Reg. No./Substance:
0/Biological Factors; 0/Cyclooxygenase Inhibitors; 0/Potassium Channel Blockers; 0/Prostaglandins; 0/endothelium-dependent hyperpolarization factor; 10102-43-9/Nitric Oxide; 115422-61-2/Charybdotoxin; 2149-70-4/Nitroarginine; 24345-16-2/Apamin; 53-86-1/Indomethacin; EC 1.14.13.39/Nitric Oxide Synthase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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