Document Detail


The effects of ascorbic acid on dopamine-induced death of PC12 cells are dependent on exposure kinetics.
MedLine Citation:
PMID:  11108986     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The role of ascorbic acid on dopamine (DA) oxidation-mediated cytotoxicity was studied using the PC12 cell line. DA cytotoxicity was slightly attenuated by ascorbic acid, whereas the cytotoxicity of 6-hydroxydopamine (6-OHDA), a DA oxidation product, was markedly potentiated. To elucidate the relationship between the ascorbic acid effect and the degree of DA oxidation, ascorbic acid was added in a time-dependent fashion after DA treatment. We found greater cell death the later ascorbic acid was applied. Treatment of cells with glutathione alleviated DA- and 6-OHDA-induced cell death, while L-buthionine sulfoximine potentiated DA and 6-OHDA cytotoxicity. Ascorbic acid combined with glutathione eliminated the toxicity of DA and 6-OHDA. These results suggest that the interaction between DA and ascorbic acid is dependent upon the degree of DA oxidation and glutathione availability.
Authors:
H Y Choi; J H Song; D K Park; G M Ross
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Neuroscience letters     Volume:  296     ISSN:  0304-3940     ISO Abbreviation:  Neurosci. Lett.     Publication Date:  2000 Dec 
Date Detail:
Created Date:  2000-12-28     Completed Date:  2001-02-15     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  7600130     Medline TA:  Neurosci Lett     Country:  IRELAND    
Other Details:
Languages:  eng     Pagination:  81-4     Citation Subset:  IM    
Affiliation:
Department of Physiology, Queen's University, Kingston, K7L 3N6, Ontario, Canada.
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MeSH Terms
Descriptor/Qualifier:
Animals
Ascorbic Acid / metabolism,  pharmacology*
Buthionine Sulfoximine / pharmacology
Dopamine / metabolism,  toxicity*
Dose-Response Relationship, Drug
Drug Interactions / physiology*
Glutathione / metabolism,  pharmacology
Nerve Degeneration / chemically induced*,  metabolism,  physiopathology
Neuroprotective Agents / metabolism,  pharmacology
Neurotoxins / metabolism,  pharmacology*
Oxidative Stress / drug effects*,  physiology
Oxidopamine / toxicity
PC12 Cells / cytology,  drug effects*,  metabolism
Parkinson Disease / etiology,  metabolism,  physiopathology
Pharmacokinetics
Rats
Chemical
Reg. No./Substance:
0/Neuroprotective Agents; 0/Neurotoxins; 1199-18-4/Oxidopamine; 50-81-7/Ascorbic Acid; 5072-26-4/Buthionine Sulfoximine; 70-18-8/Glutathione

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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