Document Detail


The effect of disrupting the human magnocellular pathway on global motion perception.
MedLine Citation:
PMID:  15358070     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The purpose of this study was to demonstrate the effect of human magnocellular (M)-pathway disruption on global motion perception. Coherence thresholds for global motion direction discrimination in random dot patterns were determined at slow and moderate dot speeds: (1) after adaptation to full-field sinusoidal flicker or a steady gray field, and (2) on a red or a gray background. Adaptation to flicker and a red background increased motion coherence thresholds relative to the gray baseline conditions at both dot speeds. Physiological studies have shown that M cells in the retina and LGN are inhibited by red light and are a main contributor to flicker perception in monkeys. Therefore, our results suggest that interference with processing in the subcortical M pathway disrupts higher-level motion integration.
Authors:
Craig Chapman; Ryan Hoag; Deborah Giaschi
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Vision research     Volume:  44     ISSN:  0042-6989     ISO Abbreviation:  Vision Res.     Publication Date:  2004  
Date Detail:
Created Date:  2004-09-10     Completed Date:  2005-01-18     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0417402     Medline TA:  Vision Res     Country:  England    
Other Details:
Languages:  eng     Pagination:  2551-7     Citation Subset:  IM    
Copyright Information:
Copyright 2004 Elsevier Ltd.
Affiliation:
Department of Ophthalmology and Visual Sciences, University of British Columbia, 4480 Oak Street, Room A146, Vancouver, BC, Canada V6H 3V4.
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Discrimination (Psychology) / physiology
Female
Humans
Male
Motion Perception / physiology*
Pattern Recognition, Visual / physiology
Photic Stimulation / methods
Psychophysics
Sensory Thresholds
Visual Pathways / physiology*

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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