Document Detail


The effect of amphiphilic siloxane oligomers on fibroblast and keratinocyte proliferation and apoptosis.
MedLine Citation:
PMID:  20725963     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The formation of hypertrophic scars (HSF) is a frequent medical outcome of wound repair and often requires further therapy with treatments such as silicone gel sheets (SGS) or apoptosis-inducing agents, including bleomycin. Although widely used, knowledge regarding SGS and their mode of action is limited. Preliminary research has shown that small amounts of amphiphilic silicone present in SGS have the ability to move into skin during treatment. We demonstrate herein that a commercially available analogue of these amphiphilic siloxane species, the rake copolymer GP226, decreases collagen synthesis on exposure to cultures of fibroblasts derived from HSF. By size exclusion chromatography, GP226 was found to be a mixture of siloxane species, containing five fractions of different molecular weight. By studies of collagen production, cell viability and proliferation, it was revealed that a low molecular weight fraction (fraction IV) was the most active, reducing the number of viable cells present after treatment and thereby reducing collagen production as a result. On exposure of fraction IV to human keratinocytes, viability and proliferation were also significantly affected. HSF undergoing apoptosis after application of fraction IV were also detected via real-time microscopy and by using the TUNEL assay. Taken together, these data suggests that these amphiphilic siloxanes could be potential non-invasive substitutes to apoptotic-inducing chemical agents that are currently used as scar treatments.
Authors:
Emily C Lynam; Yan Xie; Bree Loli; Tim R Dargaville; David I Leavesley; Graeme A George; Zee Upton
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Publication Detail:
Type:  Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of biomedical materials research. Part A     Volume:  95     ISSN:  1552-4965     ISO Abbreviation:  J Biomed Mater Res A     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-09-22     Completed Date:  2011-01-13     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101234237     Medline TA:  J Biomed Mater Res A     Country:  United States    
Other Details:
Languages:  eng     Pagination:  620-31     Citation Subset:  IM    
Affiliation:
Tissue Repair and Regeneration Program, Institute of Health and Biomedical Innovation, Queensland University of Technology, Kelvin Grove, Queensland, Australia. emily.lynam@qut.edu.au
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MeSH Terms
Descriptor/Qualifier:
Apoptosis / drug effects*
Biocompatible Materials / chemistry,  pharmacology
Cell Proliferation / drug effects*
Cell Survival / drug effects
Cells, Cultured
Cicatrix, Hypertrophic / pathology
Coculture Techniques
Fibroblasts* / cytology,  drug effects,  physiology
Humans
Keratinocytes* / cytology,  drug effects,  physiology
Materials Testing
Siloxanes / pharmacology*
Surface-Active Agents / pharmacology*
Chemical
Reg. No./Substance:
0/Biocompatible Materials; 0/Siloxanes; 0/Surface-Active Agents

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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