Document Detail


A comparison of some of the pharmacological properties of the new eburnamenine derivative vindeburnol with those of vincamine, vinburnine, dihydroergotoxine mesilate and nicergoline.
MedLine Citation:
PMID:  3814205     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The effects of a new eburnamenine derivative (3 beta,14 alpha, 16 alpha)-(+/-)-14,15-dihydro-20,21-dinoreburnamenin-14-ol (vindeburnol, RU 24722) on EEG, on brain energy metabolism and on local cerebral blood flow (LCBF) and in different experimental models of cerebral insufficiency were compared with those of vincamine, vinburnine (1-eburnamonine), dihydroergotoxine mesilate and nicergoline. Vindeburnol at 2 mg/kg i.v., increased the EEG resistance time in rats subjected to asphyxia anoxia and at 10 mg/kg s.c., significantly improved the electrocortical recovery of gerbils subjected to a 10-min cerebral ischemia. Vindeburnol (10 mg/kg i.p.) significantly retarded glucose, phosphocreatine and adenosine triphosphate utilization and lactate production in mouse brain during 10 s of decapitation ischemia. The cerebral metabolic rate was 10.34 mmol/kg/min, which was about 50% of the control value. At 10 mg/kg i.p., the product induced a slight and transient increase in LCBF. Vincamine improved the early phase of the postischemic electrocortical recovery in the gerbil, had no effect on cerebral energy substrates and slightly increased the LCBF for 15 min. Dihydroergotoxine mesilate improved the early phase of the electrocortical recovery in gerbils subjected to ischemia, did not significantly modify the energy substrates and rapidly increased the LCBF, which was normal after 30 min. Vinburnine and nicergoline were inactive in the cerebral insufficiency models used and did not significantly modify cerebral energy metabolism. These results show that vindeburnol has a different pharmacological profile from vincamine, vinburnine, dihydroergotoxine mesilate and nicergoline, and suggest that vindeburnol may be therapeutically effective in cerebral insufficiency.
Authors:
F Barzaghi; M Dragonetti; M L Formento; C Gueniau; A Nencioni; P Mantegazza
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Publication Detail:
Type:  Comparative Study; Journal Article    
Journal Detail:
Title:  Arzneimittel-Forschung     Volume:  36     ISSN:  0004-4172     ISO Abbreviation:  Arzneimittelforschung     Publication Date:  1986 Oct 
Date Detail:
Created Date:  1987-03-20     Completed Date:  1987-03-20     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0372660     Medline TA:  Arzneimittelforschung     Country:  GERMANY, WEST    
Other Details:
Languages:  eng     Pagination:  1442-8     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Animals
Brain Ischemia / drug therapy
Cerebral Cortex / blood supply,  drug effects
Cerebrovascular Circulation / drug effects*
Cerebrovascular Disorders / drug therapy
Dihydroergotoxine / pharmacology*
Electroencephalography
Ergolines / pharmacology*
Gerbillinae
Male
Mice
Mice, Inbred Strains
Nicergoline / pharmacology*
Rats
Rats, Inbred Strains
Vinca Alkaloids / pharmacology*
Vincamine / analogs & derivatives,  pharmacology*
Chemical
Reg. No./Substance:
0/Ergolines; 0/Vinca Alkaloids; 11032-41-0/Dihydroergotoxine; 1617-90-9/Vincamine; 27848-84-6/Nicergoline; 474-00-0/eburnamonine; 68779-67-9/vindeburnol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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