Document Detail


The clinical pharmacology of angiotensin converting enzyme inhibitors in chronic heart failure.
MedLine Citation:
PMID:  1641405     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
ACE inhibitors (ACEIs) have now been shown to improve symptoms and survival in patients with mild, moderate and severe chronic heart failure. Their mechanism of action is thought to be a combination of RAAS suppression and augmentation of bradykinin and prostaglandins. Although ACE inhibitors improve hemodynamics post myocardial infarction, we do not yet have consistent data on their effects on symptoms or survival in these particular patients. One other potential benefit is their effects on reperfusion injury and free radicals. As yet only minor differences have been found to exist between different ACEIs but increasing attention is now being focussed in this direction.
Authors:
A D Struthers
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Pharmacology & therapeutics     Volume:  53     ISSN:  0163-7258     ISO Abbreviation:  Pharmacol. Ther.     Publication Date:  1992  
Date Detail:
Created Date:  1992-09-03     Completed Date:  1992-09-03     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  7905840     Medline TA:  Pharmacol Ther     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  187-97     Citation Subset:  IM    
Affiliation:
Department of Pharmacology and Clinical Pharmacology, Ninewells Hospital and Medical School, Dundee, Scotland, U.K.
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MeSH Terms
Descriptor/Qualifier:
Angiotensin-Converting Enzyme Inhibitors / adverse effects,  pharmacokinetics,  therapeutic use*
Biological Availability
Heart Failure / drug therapy*
Hemodynamics / drug effects
Humans
Myocardial Infarction / drug therapy
Myocardial Reperfusion Injury / drug therapy
Chemical
Reg. No./Substance:
0/Angiotensin-Converting Enzyme Inhibitors

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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