Document Detail


Is cell aging caused by respiration-dependent injury to the mitochondrial genome?
MedLine Citation:
PMID:  7037547     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Though intrinsic mitochondrial aging has been considered before as a possible cause of cellular senescence, the mechanisms of such mitochondrial aging have remained obscure. In this article we expand on our hypothesis of free-radical-induced inhibition of mitochondrial replenishment in fixed postmitotic cells. We maintain that the respiration-dependent production of superoxide and hydroxyl radicals may not be fully counteracted, leading to a continuous production of lipoperoxides and malonaldehyde in actively respiring mitochondria. These compounds, in turn, can easily react with the mitochondrial DNA which is in close spatial relationship with the inner mitochondrial membrane, producing an injury that the mitochondria may be unable to counteract because of their apparent lack of adequate repair mechanisms. Mitochondrial division may thus be inhibited leading to age-related reduction of mitochondrial numbers, a deficit in energy production with a concomitant decrease in protein synthesis, deterioration of physiological performance, and therefore, of organismic performance.
Authors:
J E Fleming; J Miquel; S F Cottrell; L S Yengoyan; A C Economos
Related Documents :
12054927 - Production of mitochondrial dna transgenic mice using zygotes.
19095717 - Mechanisms of formation and accumulation of mitochondrial dna deletions in aging neurons.
20556877 - Therapeutic prospects for mitochondrial disease.
10601867 - The d-loop structure of human mtdna is destabilized directly by 1-methyl-4-phenylpyridi...
9740127 - Specific site selection in rna resulting from a combination of nonspecific secondary st...
4017747 - Mass characteristics of dna obtained from chromosomes of a human carcinoma cell line.
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Gerontology     Volume:  28     ISSN:  0304-324X     ISO Abbreviation:  Gerontology     Publication Date:  1982  
Date Detail:
Created Date:  1982-05-27     Completed Date:  1982-05-27     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  7601655     Medline TA:  Gerontology     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  44-53     Citation Subset:  IM; S    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Cell Survival*
DNA, Mitochondrial / physiology*
Free Radicals
Lipid Peroxides / physiology
Malondialdehyde
Oxygen Consumption*
Chemical
Reg. No./Substance:
0/DNA, Mitochondrial; 0/Free Radicals; 0/Lipid Peroxides; 542-78-9/Malondialdehyde

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Mobilization of Vibrio cholerae chromosomal genes by plasmid RP4::Mu cts62
Next Document:  Hydergine in senile mental impairment.