Document Detail


c-Jun N-terminal kinase/c-Jun inhibits fibroblast proliferation by negatively regulating the levels of stathmin/oncoprotein 18.
MedLine Citation:
PMID:  20594188     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The JNKs (c-Jun N-terminal kinases) are stress-activated serine/threonine kinases that can regulate both cell death and cell proliferation. We have developed a cell system to control JNK re-expression at physiological levels in JNK1/2-null MEFs (murine embryonic fibroblasts). JNK re-expression restored basal and stress-activated phosphorylation of the c-Jun transcription factor and attenuated cellular proliferation with increased cells in G1/S-phase of the cell cycle. To explore JNK actions to regulate cell proliferation, we evaluated a role for the cytosolic protein, STMN (stathmin)/Op18 (oncoprotein 18). STMN, up-regulated in a range of cancer types, plays a crucial role in the control of cell division through its regulation of microtubule dynamics of the mitotic spindle. In JNK1/2-null or c-Jun-null MEFs or cells treated with c-Jun siRNA (small interfering RNA), STMN levels were significantly increased. Furthermore, a requirement for JNK/cJun signalling was demonstrated by expression of wild-type c-Jun, but not a phosphorylation-defective c-Jun mutant, being sufficient to down-regulate STMN. Critically, shRNA (small hairpin RNA)-directed STMN down-regulation in JNK1/2-null MEFs attenuated proliferation. Thus JNK/c-Jun regulation of STMN levels provides a novel pathway in regulation of cell proliferation with important implications for understanding the actions of JNK as a physiological regulator of the cell cycle and tumour suppressor protein.
Authors:
Yvonne Y C Yeap; Ivan H W Ng; Bahareh Badrian; Tuong-Vi Nguyen; Yan Y Yip; Amardeep S Dhillon; Steven E Mutsaers; John Silke; Marie A Bogoyevitch; Dominic C H Ng
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  The Biochemical journal     Volume:  430     ISSN:  1470-8728     ISO Abbreviation:  Biochem. J.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-13     Completed Date:  2010-09-07     Revised Date:  2010-09-22    
Medline Journal Info:
Nlm Unique ID:  2984726R     Medline TA:  Biochem J     Country:  England    
Other Details:
Languages:  eng     Pagination:  345-54     Citation Subset:  IM    
Affiliation:
Department of Biochemistry and Molecular Biology, Bio21 Institute, University of Melbourne, VIC, Australia.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Proliferation*
Cells, Cultured
Down-Regulation*
Fibroblasts / cytology*,  enzymology,  metabolism
Mice
Mice, Knockout
Mitogen-Activated Protein Kinase 8 / genetics,  metabolism*
Mitogen-Activated Protein Kinase 9 / genetics,  metabolism*
Phosphorylation
Proto-Oncogene Proteins c-jun / genetics,  metabolism*
Stathmin / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/Proto-Oncogene Proteins c-jun; 0/Stathmin; EC 2.7.1.24/Mitogen-Activated Protein Kinase 9; EC 2.7.11.24/Mitogen-Activated Protein Kinase 8

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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