Document Detail


c-Jun/AP-1 pathway-mediated cyclin D1 expression participates in low dose arsenite-induced transformation in mouse epidermal JB6 Cl41 cells.
MedLine Citation:
PMID:  19059425     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Arsenic is a well-documented human carcinogen associated with skin carcinogenesis. Our previous work reveals that arsenite exposure is able to induce cell transformation in mouse epidermal cell JB6 Cl41 through the activation of ERK, rather than JNK pathway. Our current studies further evaluate downstream pathway in low dose arsenite-induced cell transformation in JB6 Cl41 cells. Our results showed that treatment of cells with low dose arsenite induced activation of c-Jun/AP-1 pathway, and ectopic expression of dominant negative mutant of c-Jun (TAM67) blocked arsenite-induced transformation. Furthermore, our data indicated that cyclin D1 was an important downstream molecule involved in c-Jun/AP-1-mediated cell transformation upon low dose arsenite exposure, because inhibition of cyclin D1 expression by its specific siRNA in the JB6 Cl41 cells resulted in impairment of anchorage-independent growth of cells induced by low dose arsenite. Collectively, our results demonstrate that c-Jun/AP-1-mediated cyclin D1 expression is at least one of the key events implicated in cell transformation upon low dose arsenite exposure.
Authors:
Dongyun Zhang; Jingxia Li; Jimin Gao; Chuanshu Huang
Related Documents :
10502295 - 1,25-dihydroxyvitamin d(3)-induced retardation of the g(2)/m traverse is associated wit...
20811815 - (e)-1-(3,4-dihydroxyphenethyl)-3-styrylurea inhibits proliferation of mcf-7 cells throu...
19107355 - Effects of betulinic acid on proliferation and apoptosis in jurkat cells and its in vit...
15643065 - Cyclin cln3p links g1 progression to hyphal and pseudohyphal development in candida alb...
21544335 - Cell cycle dependence of drug initiated apoptosis in hl-60 cells.
3088215 - Modulation in vitro of immune parameters in homosexual males with the preclinical compl...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2008-11-14
Journal Detail:
Title:  Toxicology and applied pharmacology     Volume:  235     ISSN:  1096-0333     ISO Abbreviation:  Toxicol. Appl. Pharmacol.     Publication Date:  2009 Feb 
Date Detail:
Created Date:  2009-02-09     Completed Date:  2009-03-06     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  0416575     Medline TA:  Toxicol Appl Pharmacol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  18-24     Citation Subset:  IM    
Affiliation:
Nelson Institute of Environmental Medicine, New York University School of Medicine, 57 Old Forge Road, Tuxedo, NY 10987, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Arsenites / administration & dosage,  metabolism*,  toxicity*
Cell Line
Cyclin D1 / genetics,  metabolism*
Dose-Response Relationship, Drug
Environmental Pollutants / metabolism,  toxicity
Epidermis / cytology*
JNK Mitogen-Activated Protein Kinases / metabolism*
Mice
Grant Support
ID/Acronym/Agency:
CA094964/CA/NCI NIH HHS; CA112557/CA/NCI NIH HHS; ES000260/ES/NIEHS NIH HHS; ES012451/ES/NIEHS NIH HHS
Chemical
Reg. No./Substance:
0/Arsenites; 0/Ccnd1 protein, mouse; 0/Environmental Pollutants; 136601-57-5/Cyclin D1; 15502-74-6/arsenite; EC 2.7.11.24/JNK Mitogen-Activated Protein Kinases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Doping in sport: 3. Metabolic conversion of oral norethisterone to urinary 19-norandrosterone.
Next Document:  C-type lectin protein isoforms of Macrovipera lebetina: cDNA cloning and genetic diversity.