Document Detail


The bright side of being blue: depression as an adaptation for analyzing complex problems.
MedLine Citation:
PMID:  19618990     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Depression is the primary emotional condition for which help is sought. Depressed people often report persistent rumination, which involves analysis, and complex social problems in their lives. Analysis is often a useful approach for solving complex problems, but it requires slow, sustained processing, so disruption would interfere with problem solving. The analytical rumination hypothesis proposes that depression is an evolved response to complex problems, whose function is to minimize disruption and sustain analysis of those problems by (a) giving the triggering problem prioritized access to processing resources, (b) reducing the desire to engage in distracting activities (anhedonia), and (c) producing psychomotor changes that reduce exposure to distracting stimuli. As processing resources are limited, sustained analysis of the triggering problem reduces the ability to concentrate on other things. The hypothesis is supported by evidence from many levels-genes, neurotransmitters and their receptors, neurophysiology, neuroanatomy, neuroenergetics, pharmacology, cognition, behavior, and efficacy of treatments. In addition, the hypothesis provides explanations for puzzling findings in the depression literature, challenges the belief that serotonin transmission is low in depression, and has implications for treatment.
Authors:
Paul W Andrews; J Anderson Thomson
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review    
Journal Detail:
Title:  Psychological review     Volume:  116     ISSN:  0033-295X     ISO Abbreviation:  Psychol Rev     Publication Date:  2009 Jul 
Date Detail:
Created Date:  2009-07-21     Completed Date:  2009-09-28     Revised Date:  2011-04-01    
Medline Journal Info:
Nlm Unique ID:  0376476     Medline TA:  Psychol Rev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  620-54     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2009 APA, all rights reserved.
Affiliation:
Virginia Institute for Psychiatric and Behavioral Genetics, Virginia Commonwealth University, Richmond, VA 23298-0126, USA. pandrews@vcu.edu
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Adaptation, Psychological* / physiology
Affect / physiology
Antidepressive Agents / adverse effects,  therapeutic use
Arousal / physiology
Attention* / physiology
Awareness / physiology
Biological Evolution
Depression / physiopathology,  psychology*,  therapy
Depressive Disorder, Major / physiopathology,  psychology,  therapy
Humans
Memory, Short-Term / physiology
Models, Psychological
Prefrontal Cortex / physiology
Problem Solving* / physiology
Psychomotor Performance / physiology
Psychotherapy
Serotonin / physiology
Stress, Psychological / complications,  physiopathology
Synaptic Transmission / physiology
Thinking / physiology
Grant Support
ID/Acronym/Agency:
DA-018673/DA/NIDA NIH HHS; MH-20030/MH/NIMH NIH HHS; T32 MH020030-01/MH/NIMH NIH HHS
Chemical
Reg. No./Substance:
0/Antidepressive Agents; 50-67-9/Serotonin
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  An additive-utility model of delay discounting.
Next Document:  The nature of psychological thresholds.