Document Detail


The antiinflammatory effect of dopamine in alcoholic hemorrhagic pancreatitis in cats. Studies on the receptors and mechanisms of action.
MedLine Citation:
PMID:  1659548     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hemorrhagic pancreatitis was induced in cats by perfusing pancreatic enzymes through a pancreatic duct after the administration of intragastric ethanol. Dimethyl prostaglandin E2 was administered concurrently. In the first study, dopamine's antiinflammatory effect on the pancreas was determined in the presence of haloperidol, propranolol, or both. Next, dopamine's effects on blood flow in the normal and inflamed pancreas were compared using a hydrogen gas-clearance technique. In the final study, the effect of dopamine on fluorescein isothiocyanate-labeled dextran leakage from the pancreatic duct to portal venous blood was investigated. It was found that blockade of either dopamine or beta-adrenergic receptors reduced, and blockade of both receptors completely eliminated, the antiinflammatory effect. Dopamine had no effect on pancreatic blood flow in normal cats. In pancreatitis, although dopamine transiently reduced blood flow, after an hour flow had returned to normal. Dopamine reversed the leakage of fluorescein isothiocyanate-labeled dextran from the pancreatic duct caused by ethanol and by ethanol and prostaglandin E2. It was concluded that dopamine ameliorated pancreatitis by reducing pancreatic ductal and/or microvascular permeability rather than by altering pancreatic blood flow. The antiinflammatory effect was mediated by both dopamine and beta-adrenergic receptors.
Authors:
N D Karanjia; A L Widdison; F J Lutrin; Y B Chang; H A Reber
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Gastroenterology     Volume:  101     ISSN:  0016-5085     ISO Abbreviation:  Gastroenterology     Publication Date:  1991 Dec 
Date Detail:
Created Date:  1992-01-02     Completed Date:  1992-01-02     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0374630     Medline TA:  Gastroenterology     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1635-41     Citation Subset:  AIM; IM    
Affiliation:
Department of Surgery, Veterans Administration Medical Center, Sepulveda, California.
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Alcoholism / complications*
Animals
Capillary Permeability / drug effects*
Cats
Dopamine / pharmacology,  therapeutic use*
Ethanol / pharmacology
Haloperidol / pharmacology
Hemorrhage / drug therapy*,  etiology,  pathology
Pancreas / blood supply,  drug effects*,  pathology
Pancreatitis / drug therapy*,  etiology,  pathology
Propranolol / pharmacology
Prostaglandins E / pharmacology
Receptors, Adrenergic, beta / drug effects,  immunology
Receptors, Dopamine / drug effects,  immunology
Grant Support
ID/Acronym/Agency:
DK 37553-03/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Prostaglandins E; 0/Receptors, Adrenergic, beta; 0/Receptors, Dopamine; 52-86-8/Haloperidol; 525-66-6/Propranolol; 64-17-5/Ethanol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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