Document Detail


An alternative pathway mediates the mouse and human cone visual cycle.
MedLine Citation:
PMID:  19781940     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
One of the fundamental mysteries of the human visual system is the continuous function of cone photoreceptors in bright daylight. As visual pigment is destroyed, or bleached, by light, cones require its rapid regeneration, which in turn involves rapid recycling of the pigment's chromophore. The canonical visual cycle for rod and cone pigments involves recycling of their chromophore from all-trans retinol to 11-cis retinal in the pigment epithelium, adjacent to photoreceptors. However, shortcomings of this pathway indicate the function of a second, cone-specific, mechanism for chromophore recycling. Indeed, biochemical and physiological studies on lower species have described a cone-specific visual cycle in addition to the long-known pigment epithelium pathway. Two important questions remain, however: what is the role of this pathway in the function of mammalian cones, and is it present in higher mammals, including humans? Here, we show that mouse, primate, and human neural retinas promote pigment regeneration and dark adaptation selectively in cones, but not in rods. This pathway supports rapid dark adaptation of mammalian cones and extends their dynamic range in background light independently of the pigment epithelium. This pigment-regeneration mechanism is essential for our daytime vision and appears to be evolutionarily conserved.
Authors:
Jin-Shan Wang; Vladimir J Kefalov
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2009-09-24
Journal Detail:
Title:  Current biology : CB     Volume:  19     ISSN:  1879-0445     ISO Abbreviation:  Curr. Biol.     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-10-14     Completed Date:  2010-03-08     Revised Date:  2010-12-03    
Medline Journal Info:
Nlm Unique ID:  9107782     Medline TA:  Curr Biol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1665-9     Citation Subset:  IM    
Affiliation:
Department of Ophthalmology and Visual Sciences, Washington University School of Medicine, St. Louis, MO 63110, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Dark Adaptation / physiology*
Electroretinography
Humans
Light / adverse effects
Mice
Retinal Cone Photoreceptor Cells / physiology*,  radiation effects
Retinal Pigments / metabolism*
Vision, Ocular / physiology*
Vitamin A / metabolism
Grant Support
ID/Acronym/Agency:
EY 019312/EY/NEI NIH HHS; EY 02687/EY/NEI NIH HHS; R01 EY019312-01/EY/NEI NIH HHS
Chemical
Reg. No./Substance:
0/Retinal Pigments; 11103-57-4/Vitamin A

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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