Document Detail


alphaB-crystallin suppresses oxidative stress-induced astrocyte apoptosis by inhibiting caspase-3 activation.
MedLine Citation:
PMID:  19379782     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
alphaB-crystallin is a member of the small heat shock proteins, which is abundantly expressed in various vertebrate tissues including the central nervous system. In our previous report, we showed alphaB-crystallin induction in activated astrocytes in the postischemic brain and in H2O2-treated primary astrocyte cultures. To investigate the functional significance of alphaB-crystallin induction in astrocytes, we generated a stable C6 astroglioma cell line overexpressing alphaB-crystallin. In these cells, hydrogen peroxide-induced apoptosis was reduced by 60% compared to parent cells. Furthermore, the repression of alphaB-crystallin expression by alphaB-crystallin siRNA transfection suppressed this protective effect, indicating that alphaB-crystallin is responsible for the protection against H2O2-induced apoptosis in C6 astroglioma cells. Similar level of aggravation in H2O2-induced apoptosis was observed in primary astrocyte cultures when alphaB-crystallin expression was suppressed by alphaB-crystallin siRNA transfection, confirming the importance of alphaB-crystallin. In addition, the induction of caspase-3 activity after H2O2 treatment was markedly suppressed in alphaB-crystallin-overexpressing cells, and immunoprecipitation proved binding between alphaB-crystallin and partially processed caspase-3 (a p24 intermediate). These results indicate that alphaB-crystallin confers protection against hydrogen peroxide-induced astrocytes apoptosis in part by inhibiting caspase-3 activation.
Authors:
Joo-Hyun Shin; Seung-Woo Kim; Chae-Moon Lim; Ji-Young Jeong; Chun-Shu Piao; Ja-Kyeong Lee
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-04-18
Journal Detail:
Title:  Neuroscience research     Volume:  64     ISSN:  1872-8111     ISO Abbreviation:  Neurosci. Res.     Publication Date:  2009 Aug 
Date Detail:
Created Date:  2009-06-16     Completed Date:  2009-12-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8500749     Medline TA:  Neurosci Res     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  355-61     Citation Subset:  IM    
Affiliation:
Department of Anatomy, Inha University School of Medicine, Inchon, Republic of Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects,  physiology*
Astrocytes / drug effects,  metabolism*
Caspase 3 / metabolism*
Cell Line, Tumor
Cytoprotection / physiology
Down-Regulation / physiology
Hydrogen Peroxide / toxicity
Neurodegenerative Diseases / genetics,  metabolism,  physiopathology
Oxidants / toxicity
Oxidative Stress / drug effects,  physiology*
Protein Binding / physiology
RNA Interference / physiology
Rats
Transfection
alpha-Crystallin B Chain / genetics*,  metabolism*
Chemical
Reg. No./Substance:
0/Oxidants; 0/alpha-Crystallin B Chain; 7722-84-1/Hydrogen Peroxide; EC 3.4.22.-/Caspase 3

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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