Document Detail


The aged epidermal permeability barrier: basis for functional abnormalities.
MedLine Citation:
PMID:  11913735     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Aged epidermis develops an abnormality in permeability barrier homeostasis, which is accentuated further in photoaged skin. The biochemical basis is a global reduction in stratum corneum lipids and profound abnormality in cholesterol synthesis. Various cytokine/growth factor signaling pathways are abnormal in aged skin, particularly in the interleukin-1 family. Barrier repair therapy can be effective in restoring normal function if a cholesterol-dominant mixture of the three key physiologic lipids, including ceramides and free fatty acids, is emphasized.
Authors:
Peter M Elias; Ruby Ghadially
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Clinics in geriatric medicine     Volume:  18     ISSN:  0749-0690     ISO Abbreviation:  Clin. Geriatr. Med.     Publication Date:  2002 Feb 
Date Detail:
Created Date:  2002-03-26     Completed Date:  2002-05-10     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  8603766     Medline TA:  Clin Geriatr Med     Country:  United States    
Other Details:
Languages:  eng     Pagination:  103-20, vii     Citation Subset:  IM    
Affiliation:
Department of Dermatology, University of California, San Francisco, USA. eliaspm@itsa.ucsf.edu
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MeSH Terms
Descriptor/Qualifier:
Aged
Aging / physiology
CCAAT-Enhancer-Binding Proteins / physiology
Cell Membrane Permeability*
DNA-Binding Proteins / physiology
Epidermal Growth Factor / physiology
Epidermis / physiology*
Humans
Interleukin-1 / physiology
Skin Aging / physiology
Sterol Regulatory Element Binding Protein 1
Transcription Factors / physiology
Chemical
Reg. No./Substance:
0/CCAAT-Enhancer-Binding Proteins; 0/DNA-Binding Proteins; 0/Interleukin-1; 0/SREBF1 protein, human; 0/Sterol Regulatory Element Binding Protein 1; 0/Transcription Factors; 62229-50-9/Epidermal Growth Factor

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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