Document Detail


An acute model for IgA-mediated glomerular inflammation in rats induced by monoclonal polymeric rat IgA antibodies.
MedLine Citation:
PMID:  8099859     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
An acute model for IgA-mediated glomerular inflammation in rats was induced by the in situ deposition of IgA directly into the glomerular mesangium. F(ab')2 anti-Thy1 MoAb was used to anchor an antigen, DNP (2,4-dinitrophenol), in the glomeruli of rats. Subsequent infusion of rat polymeric (p-) or monomeric (m-) IgA MoAb with specificity for DNP resulted in mesangial deposition of IgA in both groups of rats. However, acute proteinuria was observed only in p-IgA-treated rats and not in PBS- or m-IgA-treated rats. Immunofluorescence analysis revealed deposition of C3 in an identical pattern to that of IgA in the glomeruli of p-IgA-treated rats. No mesangial deposits of C4 or C1q were seen in these animals. Rats receiving m-IgA or PBS displayed no detectable C3, C4 or C1q deposition. The amount of proteinuria in p-IgA-treated rats was related to the amount of deposited C3. The presence of intraglomerular monocytes was only observed 2 days after p-IgA injection. By light microscopy, aneurysm formation, mesangial hypercellularity and matrix expansion were seen only in p-IgA-treated rats. However, by 37 days post-injection complete resolution of the lesions was observed. No histological renal changes were observed in PBS- or m-IgA-treated rats. In conclusion, an acute form of IgA-mediated nephritis in rats was induced by p-IgA but not by m-IgA. This reproducible model provides a basis for further study into the mechanisms of IgA-mediated glomerular inflammation.
Authors:
R K Stad; J A Bruijn; D J van Gijlswijk-Janssen; L A van Es; M R Daha
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Clinical and experimental immunology     Volume:  92     ISSN:  0009-9104     ISO Abbreviation:  Clin. Exp. Immunol.     Publication Date:  1993 Jun 
Date Detail:
Created Date:  1993-07-20     Completed Date:  1993-07-20     Revised Date:  2009-11-18    
Medline Journal Info:
Nlm Unique ID:  0057202     Medline TA:  Clin Exp Immunol     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  514-21     Citation Subset:  IM    
Affiliation:
Department of Nephrology, University Hospital Leiden, The Netherlands.
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MeSH Terms
Descriptor/Qualifier:
2,4-Dinitrophenol
Acute Disease
Animals
Antibodies, Monoclonal
Antigens, Surface / immunology
Antigens, Thy-1
Complement Hemolytic Activity Assay
Complement System Proteins / immunology
Dinitrophenols / immunology
Disease Models, Animal
Fluorescent Antibody Technique
Glomerular Mesangium / immunology
Glomerulonephritis, IGA / immunology*,  pathology
Hematuria / pathology
Immunoglobulin A
Male
Membrane Glycoproteins / immunology
Polymers
Proteinuria / pathology
Rats
Rats, Wistar
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Antigens, Surface; 0/Antigens, Thy-1; 0/Dinitrophenols; 0/Immunoglobulin A; 0/Membrane Glycoproteins; 0/Polymers; 51-28-5/2,4-Dinitrophenol; 9007-36-7/Complement System Proteins
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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