Document Detail


Zinc regulates the ability of Cdc25C to activate MPF/cdk1.
MedLine Citation:
PMID:  17443687     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Zn(2+) is an essential micronutrient for the growth and development of multicellular organisms, as Zn(2+) deficiencies lead to growth retardation and congenital malformations (Vallee, BL, Falchuk, KH. 1993. Physiol Rev., 73:79-118). At the cellular level Zn(2+) depravation results in proliferation defects in many cell types (Vallee, BL, Falchuk, KH. 1993. Physiol Rev., 73:79-118), however the molecular pathways involved remain poorly defined. Here we show that the transition metal chelator TPEN (N,N,N',N'-tetrakis(2-pyridylmethyl) ethylene diamine) blocks the G2/M transition of the meiotic cell cycle by inhibiting Cdc25C-cdk1 activation. ICP-MS analyses reveal that Cdc25C is a Zn(2+)-binding metalloprotein, and that TPEN effectively strips Zn(2+) away from the enzyme. Interestingly, although apo-Cdc25C (Zn(2+)-deficient) remains fully catalytically active, it is compromised in its ability to dephosphorylate and activate MPF/cdk1. Thus, Zn(2+) is an important regulator of Cdc25C function in vivo. Because of the conserved essential role of the Cdc25C-cdk1 module in the eukaryotic cell cycle, these studies provide fundamental insights into cell cycle regulation.
Authors:
Lu Sun; Yingtao Chai; Robyn Hannigan; Venkata K Bhogaraju; Khaled Machaca
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Publication Detail:
Type:  In Vitro; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of cellular physiology     Volume:  213     ISSN:  0021-9541     ISO Abbreviation:  J. Cell. Physiol.     Publication Date:  2007 Oct 
Date Detail:
Created Date:  2007-07-26     Completed Date:  2007-10-26     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  0050222     Medline TA:  J Cell Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  98-104     Citation Subset:  IM    
Copyright Information:
(c) 2007 Wiley-Liss, Inc.
Affiliation:
Department of Physiology and Biophysics, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
CDC2 Protein Kinase / metabolism*
Cell Cycle Proteins / metabolism*
Chelating Agents / pharmacology
Enzyme Activation / drug effects
Ethylenediamines / pharmacology
Female
Maturation-Promoting Factor / metabolism*
Meiosis / drug effects
Oocytes / cytology,  drug effects,  metabolism
Phosphorylation
Recombinant Fusion Proteins / metabolism
Xenopus
Xenopus Proteins / metabolism
Zinc / metabolism*
cdc25 Phosphatases / metabolism*
Grant Support
ID/Acronym/Agency:
GM-61829/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Cell Cycle Proteins; 0/Chelating Agents; 0/Ethylenediamines; 0/Recombinant Fusion Proteins; 0/Xenopus Proteins; 16858-02-9/N,N,N',N'-tetrakis(2-pyridylmethyl)ethylenediamine; 7440-66-6/Zinc; EC 2.7.11.22/CDC2 Protein Kinase; EC 2.7.11.22/Maturation-Promoting Factor; EC 3.1.3.48/cdc25 Phosphatases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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